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Testosterone increases muscle mass regardless of DHT conversion blocking.

Dihydrotestosterone (DHT) increases oil production in skin cells by activating mTOR, and mTOR inhibitors can reduce this effect.

After prostate removal surgery, higher initial prostate size was linked to higher urethral PSA levels and more severe male pattern baldness.

Dihydrotestosterone increases certain inflammatory signals in skin cells, potentially contributing to acne.

Androgens increase growth factors in skin cells, which may lead to acne.

Free fatty acids may increase androgen production, potentially contributing to polycystic ovary syndrome.

Testosterone's muscle-building effects do not require its conversion to DHT.

Vitamin C derivative reduces hair loss-related protein in cells.

Finasteride, when taken daily, lowers dihydrotestosterone levels but doesn't noticeably affect adrenal steroid production, except by blocking the 5 alpha-reductase enzyme.

Testosterone and dihydrotestosterone play a role in women's health issues like excess hair and baldness, and treatments blocking these hormones may help.

Topical finasteride doesn't reduce DHT levels, hinting at an endocrine role in hair loss.

DHT affects hair follicle cells by changing microRNA levels, leading to less cell growth and more cell death.

Genetic defects in androgen production are linked to male developmental disorders and are improving treatment understanding.

An extract from Quercus acutissima bark was found to reduce sebum production and block an enzyme linked to acne.

Antiandrogens can treat acne, hirsutism, and seborrhea by reducing sebum production and androgen action.

Androgen affects oil production and hair growth in the skin.

Androgens like testosterone affect oil production and hair growth in skin and hair follicles.

Finasteride can have lasting negative effects on brain function and behavior by disrupting neurosteroid production.

Impaired androgen production in 46,XY DSDs causes ambiguous genitalia and requires long-term care.

Blocking DHT production more strongly may help control advanced prostate cancer and improve quality of life.

Genetic defects in testosterone production can cause hormonal and developmental disorders, and more research is needed to understand androgen regulation and develop safer treatments.

Steroidogenesis inhibitors change but don't stop androgen production in prostate cancer.

Human sebaceous glands contain enzymes that affect androgen production and may influence sebum production and acne.

Testosterone in human pancreas cells boosts insulin production.

Androgens like testosterone affect hair growth and oil production differently across body parts and individuals.

Prostate cancer can progress even with low testosterone due to internal hormone production in the tumor.

Lotus corniculatus seed extract reduces oily skin by decreasing sebum production.

Finasteride may decrease semen quality but not harm sperm production, and stopping the drug can improve semen quality; hyperprolactinemia can cause infertility but is treatable with medication.

Prostate cells have proteins that bind to specific hormones, which can increase protein production when activated by these hormones.

Dutasteride and finasteride do not significantly affect bone density, blood fats, or blood production, but slightly lower PSA levels and minimally impact sexual function in healthy young men.