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The nervous system helps control stem cell behavior and immune responses, affecting tissue repair and maintenance.

Different species and human skin models vary in their skin enzyme activities, with pig skin and some models closely matching human skin, useful for safety assessments and understanding the skin's protective roles.

Hair follicles help skin immune recovery after UVB exposure.

Different scaffold patterns improve wound healing and immune response in mouse skin, with aligned patterns being particularly effective.

Melanocytes are diverse cells important for pigmentation and skin health, influenced by genetics and environment.

Skin reactions to drugs are common and can be deadly, usually requiring stopping the drug and may be better prevented with genetic testing in the future.

PRP is effective for treating hair loss, especially with other treatments.

Men are more likely to get infectious skin diseases, while women are more prone to autoimmune and pigment-related skin conditions, influenced by biological and environmental factors.

Low-level laser therapy may help with hair regrowth in alopecia areata but its effectiveness for psoriasis and atopic dermatitis needs more research.

People with atopic dermatitis are more likely to develop other skin conditions due to shared genetics and immune pathways.

New findings suggest potential treatments for melanoma, hyperpigmentation, hair defects, and multiple sclerosis, and show skin microbiome changes don't cause atopic dermatitis.

Faulty Notch signalling may cause hair follicle changes and inflammation in hidradenitis suppurativa.

RANKL improves the immune response against herpes simplex virus by enhancing T cell activation and could help develop better treatments or vaccines.

Non-immune dermal cells dominate, epidermal cells increase after day 9, and certain immune cells persist beyond inflammation in wound-induced hair follicle regeneration.

Alopecia areata is likely an autoimmune disease with unclear triggers, involving various immune cells and molecules, and currently has no cure.

Blocking EGFR in skin cells doesn't majorly increase inflammation markers.

Some cells may slow melanoma growth, a protein could affect skin pigmentation, a gene-silencing method might treat hair defects, skin bacteria changes likely result from eczema, and a defensin protein could help treat multiple sclerosis.

The study concluded that immune cells attacking hair follicles cause hair loss in alopecia, with genetics and environment also playing a role, and highlighted the potential of certain treatments.

Antigen presenting cells around hair follicles are crucial in SLE-related hair loss.

Nitric oxide is important for skin functions and both helps protect against and contributes to skin inflammation and sensitization.

Radiation therapy damages skin structure and immune function, causing inflammation and potential hair loss.

Different parts of the body have unique immune characteristics in their skin.

Canine pemphigus foliaceus involves significant immune activity and shares similarities with human pemphigus.

Changing Wnt signaling can lead to more or less hair growth and might help treat hair loss and skin conditions.

A protein called lfTSLP is important in causing allergic and other skin diseases and could be a target for treatment.

A patient with Crohn's disease grew back their hair after stopping Adalimumab, which had caused hair loss.

Regulatory T-cells are important for healing and regenerating tissues in various organs by controlling immune responses and aiding stem cells.

Skin stem cells are maintained by signals from nearby cells and vary in their ability to renew and mature.

Blocking TSLP reduces skin inflammation and cell overgrowth in psoriasis.

The role of γδT-cells in causing alopecia areata remains unclear.