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Lack of Vitamin D receptor changes skin structure and increases certain immune cells in the skin.

Lack of Evi in skin causes psoriasis-like symptoms in mice.

BST2 protein and certain T cells increase in early alopecia areata.

Inflammation plays a key role in activating skin stem cells for hair growth and wound healing, but more research is needed to understand how it directs cell behavior.

Loss of a specific protein in skin cells causes symptoms similar to psoriasis.

A protein called EGFR protects hair follicle stem cells, and when it's disrupted, hair follicles can be damaged, but blocking certain pathways can restore hair growth.

The skin has a complex immune system that is essential for protection and healing, requiring more research for better wound treatment.

The document concludes that better targeted treatments are needed for wound healing, and single-cell technologies may improve cell-based therapies.

Leptin-deficient mice, used as a model for Type 2 Diabetes, have delayed wound healing due to impaired contraction and other dysfunctional cellular responses.

Gamma delta T cells in the skin help with healing and defense but can also cause autoimmune issues, and more research is needed to understand how they are activated.

A protein from certain immune cells is key for new hair growth after skin injury in mice.

Skin health and repair depend on the signals between skin stem cells and their surrounding cells.

Memory regulatory T cells need IL-7, not IL-2, to stay in peripheral tissues.

Skin's Regulatory T cells are crucial for maintaining skin health and could be targeted to treat immune-related skin diseases and cancer.

Certain types of T cells are essential for healthy skin and play a role in skin diseases, but more research is needed to improve treatments.

Regulatory T cells are essential for normal and improved wound healing in mice.

Special immune cells called Treg cells are important for maintaining and regenerating hair by activating a specific growth signal in hair stem cells.

Special immune cells called Regulatory T cells help control skin inflammation and repair in various skin diseases.

Human dermal γδT-cells respond to stress in hair follicles, contributing to hair loss.

Imiquimod cream activates hair follicle stem cells and causes early hair growth by changing immune cells and certain protein expressions.

Regulatory T cells help heal skin and grow hair, and their absence can lead to healing issues and hair loss.

Skin stem cells are crucial for maintaining and repairing skin, with potential for treating skin disorders and improving wound healing.

A specific group of slow-growing stem cells marked by Thy1 is crucial for skin maintenance and healing in mice.

Hair follicle development and microbes help regulatory T cells gather in newborn skin.

Activating TLR9 helps heal wounds and regrow hair by using specific immune cells.

Hair follicle-derived IL-7 and IL-15 are crucial for maintaining skin-resident memory T cells and could be targeted for treating skin diseases and lymphoma.

Stem cells help heal skin wounds by moving and changing roles, working with other cells, and needing more research on their activation and behavior.

Different immune cells like platelets, mast cells, neutrophils, macrophages, T cells, B cells, and innate lymphoid cells all play roles in skin wound healing, but more research is needed due to inconsistent results and the complex nature of the immune response.

Keratinocytes help heal skin wounds by interacting with immune cells and producing substances that kill pathogens.

Immune cells affect hair growth and could lead to new hair loss treatments.