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Aging slows wound healing due to weaker cells and immune response.

A mouse gene mutation increases the risk of skin cancer.

Certain daily habits like stress, diet, and sleep can affect the severity of hair loss in alopecia areata.

Asparagus racemosus and Withania somnifera can help reduce side effects of a cancer drug.

Leptin-deficient mice, used as a model for Type 2 Diabetes, have delayed wound healing due to impaired contraction and other dysfunctional cellular responses.

Androgens like testosterone may help treat traumatic brain injury by reducing mitochondrial stress and inflammation.

Hair dye ingredient PPD causes cell death and aging in human hair cells by altering microRNA levels.

Skin cells show flexibility in healing wounds and forming tumors, with potential for treating hair disorders and chronic ulcers.

Lung and liver macrophages protect our tissues and their dysfunction can cause various diseases.

Different types of RNAs are found in varying amounts in patients with Polycystic Ovary Syndrome, suggesting they could be important in the disease's development and potentially used as disease markers.

Enterococcus faecalis delays wound healing by disrupting cell functions and creating an anti-inflammatory environment.

Mesenchymal Stem/Stromal Cells (MSCs) help in wound healing and tissue regeneration, but can also contribute to tumor growth. They show promise in treating chronic wounds and certain burns, but their full healing mechanisms and potential challenges need further exploration.

Blocking the CXCR3 receptor reduces T cell accumulation in the skin and prevents hair loss in mice.

Certain gene variations increase the risk of alopecia areata in Koreans.

Neutrophils are important for causing chronic itch in atopic dermatitis, and blocking the CXCR3 receptor may reduce this itch.

CXCL12 protein slows down hair growth through its receptor CXCR4. Blocking this can potentially increase hair growth.

Neutrophils are key in causing chronic itch in atopic dermatitis, and blocking CXCR3 could reduce this itch.

Mice without the p21 gene can fully regenerate injured ears due to reduced Sdf1 increase and leukocyte recruitment, suggesting new ways to induce tissue regeneration in mammals.

SDF-1/CXCL12 and its receptor CXCR4 are crucial for melanocyte movement in mouse hair follicles.

The right cells and signals can potentially lead to scarless wound healing, with a mix of natural and external wound healing controllers possibly being the best way to achieve this.

Alopecia areata shows a unique type 1 interferon signature, suggesting potential treatment by targeting this pathway.

Stretching skin increases a certain protein that attracts stem cells, helping skin regeneration.

Antroquinonol may help prevent skin depigmentation by suppressing certain immune cells.

The study found that a specific genetic variation in the TNFα gene is significantly linked to Alopecia Areata in the Jordanian Arab population.

Autoimmune reactions may cause both alopecia areata and HAM.

Using a specific binding agent and low doses of FK506 can stimulate stem cells, speeding up skin healing by 25% and improving skin quality in rats and mice.

Targeting CXCL10 may help treat alopecia areata.

SFRP2 and PTGDS may be key factors in female hair loss.

The research suggests that SFRP2 and PTGDS proteins might be indicators of female pattern hair loss and could contribute to hair loss.

ILC1-like cells may contribute to hair loss in alopecia areata.