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Patients with myotonic dystrophy often have skin problems that suggest early aging and vitamin D issues, and the severity of these problems is linked to their genetic condition and vitamin D levels.

There is no cure for myotonic dystrophy type 1, so treatment focuses on managing symptoms and complications.

Myotonic Dystrophy may age cells faster, and drugs that target aging could be potential treatments.

People with myotonic dystrophy type 1 have a higher chance of getting skin tumors, including melanoma.

A man with myotonic dystrophy type 1 had 28 skin cancers, suggesting a link between the disease and skin cancer, emphasizing the need for sun protection and regular skin checks.

Rare ULBP3 gene changes may raise the risk of Alopecia areata, a certain FAS gene deletion could cause a dysfunctional protein in an immune disorder, and having one copy of a specific genetic deletion is okay, but two copies cause sickle cell disease.

People with Myotonic Dystrophy type 1 are more likely to have certain skin conditions, but not more likely to get skin cancer.

Hair follicle cells help maintain and support stem cells and blood cell formation.

Wnt1a helps keep cells that can grow hair effective for potential hair loss treatments.

Myotonic dystrophy should be considered in patients with hair thinning, and genetic counseling is important.

Scientists made a mouse that shows how a specific protein in the skin changes and affects hair growth and shape.

Women with myotonic dystrophy might get diseases related to male hormones because their body tissues are extra sensitive to these hormones.

Researchers found a new mutation causing total hair loss from birth.

Researchers isolated a new type of stem cell from mouse skin that can renew itself and turn into multiple cell types.

Cysteine-rich keratins evolved independently in mammals, reptiles, and birds for hard skin structures like hair, claws, and feathers.

The research found genetic differences in identical twins that could explain why one twin has a disease while the other does not.

Researchers created a mouse model of a type of rickets that does not cause hair loss.

A patient with Satoyoshi syndrome improved with a treatment combining several medications, including carbamazepine and methotrexate.

Childhood ptosis can vary from a minor cosmetic issue to a serious condition and may signal other health problems.

Hair and skin can regenerate without bulge stem cells due to other compensating cells.

The S250C variant in a gene may cause autoimmunity and immunodeficiency by impairing protein function.

Vav2 and Vav3 proteins help control skin stem cell numbers and activity in both healthy and cancerous cells.

PNKP is essential for keeping adult mouse progenitor cells healthy and growing normally.

Pangolins have lost some skin-related genes, but kept others, leading to their unique scales and skin features.

Increasing Rps14 helps grow more inner ear cells and repair hearing cells in baby mice.

Not having enough or having too much of the protein Grainyhead-like 3 leads to various developmental problems.

Activating β-catenin increases melanocytes and decreases Schwann cells.

A gene variant causes patched hair loss in mice, similar to alopecia areata in humans.

Certain immune cells in the skin release a protein that stops hair growth by keeping hair stem cells inactive.

CD34+ and CD34- melanocyte stem cells have different regenerative abilities.