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Anti-androgen therapy may boost immunity but increases injection site pain in vaccinated patients.

Androgenetic alopecia involves immune cell disruptions, especially increased CD4+ T cells around hair follicles.

Different immune responses cause hair loss in scalp diseases, with unique patterns in scalp psoriasis possibly protecting against hair loss.

T-cell reconstitution after thymus transplantation can cause hair whitening and loss.

Key genes linked to immune response are upregulated in hair follicles and skin tissues in chronic discoid lupus erythematosus.

Key genes linked to immune response are highly active in lupus-affected hair follicles.

Human body's immune cells are more common in the layer of fat just beneath the skin than in deeper fat layers.

Different markers are found in stem cells of the scalp's hair follicle bulge and the surrounding skin.

The document concludes that immune system abnormalities cause alopecia areata, but the exact process is still not completely understood.

The calcineurin/NFAT pathway plays a significant role in the development and growth of a type of skin cancer called cutaneous squamous cell carcinoma.

The 2015 Hair Research Congress concluded that stem cells, maraviroc, and simvastatin could potentially treat Alopecia Areata, topical minoxidil, finasteride, and steroids could treat Frontal Fibrosing Alopecia, and PTGDR2 antagonists could also treat alopecia. They also found that low-level light therapy could help with hair loss, a robotic device could assist in hair extraction, and nutrition could aid hair growth. They suggested that Alopecia Areata is an inflammatory disorder, not a single disease, indicating a need for personalized treatments.

An 11-year-old child with total hair loss may have a genetic autoimmune disease, and the outlook for hair regrowth is not good.

Higher antigliadin antibodies are linked to more severe alopecia areata, suggesting screening for celiac disease in these patients.

Higher antigliadin antibody levels are linked to alopecia areata severity.

Histone modification is key in treating chronic inflammatory skin diseases.

New protein changes may be involved in the immune attack on hair follicles in alopecia areata.

Oxidative stress plays a significant role in alopecia areata, and new treatments may include JAK inhibitors and antioxidants.

Teriflunomide helps reduce multiple sclerosis symptoms and is safe for most patients.

The extracellular matrix affects where tumors can start in the body.

The nail matrix has a reduced immune response, protecting it from autoimmunity.

Alopecia Areata is an autoimmune hair loss condition, with various treatments showing mixed effectiveness and no guaranteed cure.

The document concludes that more research is needed to find effective treatments for Lichen planopilaris and Frontal fibrosing alopecia.

Innate lymphoid cells type 1 may contribute to alopecia areata by damaging hair follicles.

Canine pemphigus foliaceus involves significant immune activity and shares similarities with human pemphigus.

Topical JAK inhibitors can effectively treat alopecia areata and vitiligo by modulating immune responses.

Regulatory T cells are essential for normal and improved wound healing in mice.

Diffuse alopecia areata involves more inflammation and higher allergy-related antibodies than patchy types.

Using fat-derived stem cells with the drug meglumine antimoniate can help control skin disease and reduce parasites in mice with leishmaniasis.

Some patients with mycosis fungoides or Sézary syndrome experience hair loss, which may be similar to alopecia areata or linked to skin lesions, possibly due to abnormal T cells, and bexarotene can help treat it.

ILC1 cells contribute to hair loss in alopecia areata.