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Neurohormones help control skin health and could treat skin disorders.

Cytotoxic T cells cause hair loss in chronic alopecia areata.

Controlled delivery of specific RNA and IL-4 restored hair growth in mice with autoimmune alopecia.

Botulinum A toxin injections reduced pain and promoted hair regrowth in a woman with a rare form of alopecia areata.

Tofacitinib may effectively treat hair loss and improve symptoms in autoimmune conditions like alopecia areata and Sjögren's syndrome, but long-term treatment might be necessary.

The protein p53 directly reduces the production of Keratin 17, a skin and hair protein, in rats with radiation dermatitis.

PCOS causes infertility mainly due to hormonal imbalances, insulin resistance, and chronic inflammation.

Increasing Treg cells in the skin does not cure hair loss from alopecia areata in mice.

Diphencyprone treatment increases CD8 lymphocytes in the scalp, which is associated with hair regrowth in alopecia areata patients.

The "color-transition sign" helps tell apart alopecia areata incognita from telogen effluvium by looking at hair color changes.

The document concludes that alopecia areata is an unpredictable autoimmune hair loss condition with no cure, but various treatments exist that require personalized approaches.

The document explains different types of hair loss, their causes, and treatments, and suggests future research areas.

Intramuscular corticosteroids are effective and safe for severe alopecia areata, with most patients regrowing hair, but nearly half may relapse.

PPAR-γ may be a key target for treating alopecia areata and other skin conditions.

Tofacitinib is safe and effective for treating moderate-to-severe alopecia areata.

Lymphocytes, especially CD8+ T cells, play a key role in causing alopecia areata, and targeting them may lead to new treatments.

ILC1-like cells can cause alopecia areata by attacking hair follicles.

Alopecia areata can be reversed by JAK inhibitors, promoting hair regrowth.

Intestinal intraepithelial lymphocytes are crucial for gut immunity and maintaining the mucosal barrier.

ILC1-like cells can independently cause alopecia areata by affecting hair follicles.

Lichen planopilaris is a chronic, scarring hair loss condition with no definitive cure, requiring accurate diagnosis and treatment to manage symptoms.

M-CSF-stimulated myeloid cells can cause alopecia areata in mice.

Teriflunomide helps reduce multiple sclerosis symptoms and is safe for most patients.

The document concludes that ongoing research using animal models is crucial for better understanding and treating Alopecia Areata.

The conditions alopecia areata, primary sclerosing cholangitis, and ulcerative colitis may be linked by shared autoimmune and cell death mechanisms.

Permanent hair dye may cause total hair loss.

Teriflunomide is an effective and generally safe oral treatment for relapsing MS, reducing relapses and slowing disability progression.

Certain immune cells contribute to severe hair loss in chronic alopecia areata, with Th17 cells possibly having a bigger impact than cytotoxic T cells.

Alopecia areata is an autoimmune disease with genetic links, treatable with certain medications, and can affect mental health.

Certain immune cells contribute to skin autoimmune diseases, and some treatments can reverse hair loss in these conditions.