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Gene therapy, especially using atoh1, shows promise for creating functional sensory hair cells in the inner ear, but dosing and side effects need to be managed for clinical application.

Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

A low dose of rapamycin increases inner ear hair cell creation by boosting SOX2+ cell numbers.

Activin A increases inner ear hair cell development, while follistatin decreases it.

Activin A promotes ear hair cell development, while follistatin delays it.

PRC2 is essential for maintaining intestinal cell balance and aiding regeneration after damage.

BMP signaling is essential for the development of touch domes.

6.1% of patients seeking PRP for hair loss had undiagnosed cicatricial alopecia, which PRP cannot treat.

An AI photographic device effectively tracked hair growth improvements in women treated for hair loss.

The niche environment controls stem cell behavior and plasticity, which is important for tissue health and repair.

Different types of stem cells with unique roles exist in blood, skin, and intestines, and this variety is important for tissue repair.

The conclusion is that tissue structure is key for stem cell communication and maintaining healthy tissues.

Skin has specialized touch receptors that can tell different sensations apart.

Using CRISPR for gene editing in the body is promising but needs better delivery methods to be more efficient and specific.

Wnt, Eda, and Shh pathways are crucial for different stages of sweat gland development in mice.

Touch sensitivity in mouse skin decreases during hair growth due to changes in touch receptors.

DNA methylation changes are linked to hair growth cycles in goats.

Epidermal stem cells have various roles in skin beyond just maintenance, including forming specialized structures and aiding in skin repair and regeneration.

Exosomes from umbilical cord cells fix hearing loss and damaged ear hair cells in mice.

Blocking adenosine A2B receptor may prevent or treat hearing loss.

Hair follicle samples effectively show how well the drug MK-0752 targets and engages with the Notch pathway.

Increasing Rps14 helps grow more inner ear cells and repair hearing cells in baby mice.

Stem cells regenerate tissues and their behavior varies by environment, suggesting the hematopoietic system model may need revision.

Hair follicles can be used to quickly assess drug effects in cancer treatment.

The conclusion is that genetic differences affect how the cochlea heals after hair cell loss, which may challenge the creation of hearing loss treatments.

Exosomes are crucial for protecting sensory hair cells in the inner ear.

Follistatin and LIN28B together improve the ability of inner ear cells in mice to regenerate into hearing cells.

Activin A and follistatin control when hair cells develop in mouse ears.

Merkel cells stabilize nerve endings in the skin, and they change independently of each other.

Activin A and follistatin control when ear hair cells form in mice.