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The vitamin D receptor is crucial for bone health and affects various body systems, with mutations potentially leading to disease.

Gata6 is important for protecting hair growth cells from DNA damage and keeping normal hair growth.

Progesterone and its byproducts control a specific receptor in the brain independently of progesterone receptors, affecting conditions related to the menstrual cycle.

The workshop highlighted the genetic links and psychological impacts of hair loss and skin disorders.

Androgens can help prevent memory problems caused by apoE4.

The balance between androgen receptor and p53 is crucial for sebaceous gland differentiation.

Hormones and their receptors, especially androgens, play a key role in hair growth and disorders like baldness.

Signals from skin cells controlled by Rac proteins help turn certain precursor cells into white fat cells.

The p75 neurotrophin receptor is important for hair follicle regression by controlling cell death.

The tumor suppressor gene FLCN affects mitochondrial function and energy use in cells.

Blocking the enzyme 5α-reductase can shrink the prostate and help treat enlarged prostate issues.

Cells in hair follicles help create fat cells in the skin by releasing a protein called Sonic Hedgehog.

miR-22, a type of microRNA, controls hair growth and its overproduction can cause hair loss, while its absence can speed up hair growth.

Pannexin 3 is important for bone formation and the development of bone cells.

The vitamin D receptor helps regulate skin and hair health independently of its usual vitamin D ligand.

Using rodents for research shows that health problems in the womb can cause diseases later in life.

Mice without the vitamin D receptor have bone issues and other health problems, suggesting vitamin D is important for preventing various diseases in humans.

Mice lacking the PPARγ gene in their fat cells had almost no fat tissue, severe metabolic problems, and abnormal development of other fat-related tissues.

Three drugs change mice's alcohol drinking patterns by affecting GABAA receptors.

Mice genetically modified to produce more CD109 in their skin had less inflammation and better healing with less scarring.

Inhibiting AP1 in mice skin causes structural changes and weakens the skin barrier.

Female mice with disrupted 5α-reductase 1 had significant metabolic issues, including stress response problems, insulin resistance, liver fat buildup, and obesity.

Damaged hair follicles make mice more prone to skin inflammation and skin cancer after UV exposure.

Dihydrotestosterone (DHT) stops hair growth in mice by lowering a growth factor important for hair.

Blocking the FGF5 gene in sheep leads to more fine wool and active hair follicles due to changes in certain cell signaling pathways.

The we/we wal/wal mice have defects in hair growth and skin layer formation, causing hair loss, useful for understanding alopecia.

Activating TRPV4 in skin cells helps regrow hair in mice, possibly offering a treatment for hair loss.

Using radiation to make mice's hair turn gray helps study and find ways to prevent or reverse hair graying.

Gamma-rays exposure during the resting phase of hair growth can damage hair regeneration and color in mice.

Blocking a specific receptor slows down hair loss in mice.