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The conclusion is that thorough investigation of hypertension and hormonal dysfunctions is important, and there may be a link between these conditions and cancer.

The study concluded that testing hormone levels after stimulation is not reliable for identifying carriers of 21-hydroxylase deficiency; genetic testing is necessary.

Mutations in the CYP21A2 gene are not a major factor in causing PCOS.

A certain genetic variation is linked to a higher risk of polycystic ovarian syndrome.

A new gene variant causes glucocorticoid resistance in a mother and son.

Most women with excess male hormones have Polycystic Ovary Syndrome, and hormonal therapy can improve symptoms but may cause side effects.

Prolactin affects hair growth cycles and can cause early hair follicle regression.

Certain mutations in the H6PD gene cause Cortisone Reductase Deficiency by affecting hormone production.

The mineralocorticoid receptor may play a role in skin and hair health and could be a new target for treating related disorders.

The L457(3.43)R mutation in the human lutropin receptor causes increased activity and hormone insensitivity, leading to precocious puberty.

Certain gene variants in estrogen receptors are linked to polycystic ovary syndrome, mainly affecting metabolism, in Tunisian women.

Mice without the vitamin D receptor have bone issues and other health problems, suggesting vitamin D is important for preventing various diseases in humans.

DHT reduces a cell's ability to promote hair growth, while 3D culture without DHT improves it.

Certain gene variants may contribute to high androgen levels in women with polycystic ovary syndrome.

Estrogen and prolactin may play bigger roles in female hair loss than previously thought.

The document concludes that Nonclassic Congenital Adrenal Hyperplasia requires personalized treatment plans to manage symptoms and fertility, with glucocorticoids being a common therapy.

The document concludes that research on sulfatase inhibitors should continue due to their potential in treating various diseases, despite some clinical trial failures.

Nonclassic congenital adrenal hyperplasia is a genetic disorder causing hormone imbalances, affecting fertility and requiring personalized treatment.

Higher Lnk protein levels in ovaries may lead to insulin resistance in women with PCOS.

The 2004 hair research meeting presented new findings on hair cell differentiation, genetic factors in hair loss, hair pigmentation, and potential targeted therapies.

The 2003 consensus updated PCOS diagnosis criteria and linked PCOS to higher risks of diabetes and heart problems, recommending lifestyle changes to lower these risks.

The document concludes that understanding hair follicle biology can lead to better hair loss treatments.

The conclusion is that proper signaling is crucial for hair growth and development, and errors can lead to cancer or hair loss.

TRP channels in the skin are important for sensation and health, and targeting them could help treat skin disorders.

Growth factors are crucial for hair development and could help treat hair diseases.

Prolactin affects the production of different keratins in human hair, which could lead to new treatments for skin and hair disorders.

The review recommends hormone replacement therapy for women with premature ovarian insufficiency to manage symptoms and protect health, with specific approaches for different groups.

The conclusion is that individual differences in COVID-19 severity are influenced by factors like age, sex, race, and genetics, which are important for personalized medicine.

Targeting glucocorticoid action might help treat type-2 diabetes, but human trials are needed.

Type 3 5α-reductase is more common and finasteride and dutasteride strongly inhibit it.