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Testing for CAG repeat polymorphism in the androgen receptor gene is not currently recommended for managing hypogonadism.

Certain gene variations and irregular X chromosome activity may contribute to hair loss in women who can have children.

E211 G>A gene linked to lower risk of severe prostate cancer and hair loss.

Certain genes and X chromosome patterns may significantly contribute to the development of hair loss.

The length of the CAG repeat in the androgen receptor gene affects the risk and progression of prostate cancer, BPH, infertility, and undermasculinized genitalia.

DHT affects bone growth by altering gene activity in osteoblasts, potentially complicating steroid use.

Shorter CAG repeats may cause hair and skin issues, while longer ones may link to acne.

Mutations in the androgen receptor gene cause Androgen Insensitivity Syndrome, affecting sexual development.

No clear link between androgen receptor variation and hair loss, but more research needed.

The study concluded that both estrogen and androgen receptors, which decrease with age, are linked to skin aging and may be hormonally regulated.

Male hormones and their receptors play a key role in hair loss and skin health, with potential new treatments being explored.

Longer CAG and GGN repeats increase alopecia risk, but no significant link to post-finasteride syndrome found.

Researchers found a gene in hamsters that responds to male hormones and may be indirectly controlled by them.

KGF and its receptor are found in enlarged prostate tissue and KGF strongly increases cell growth.

The balance between androgen receptor and p53 is crucial for sebaceous gland differentiation.

Mutations in certain receptors can cause diseases and offer new treatment options.

The androgen receptor is a promising target for breast cancer treatment, especially in triple-negative cases, but more research is needed for personalized therapies.

Prostate cancer cells can make their own androgens to activate the androgen receptor, and treatments like abiraterone may increase this ability, suggesting new therapies should target the entire steroid-making pathway.

Lowering testosterone speeds up wound healing in male mice.

A protein called CBP is found in prostate cancer and can increase the effectiveness of certain prostate cancer treatments.

Blocking the androgen receptor in skin cells reduces their growth response to male hormones, suggesting a possible treatment for skin conditions linked to androgens.

Certain genetic variants in the androgen receptor are linked to higher PSA levels, potentially affecting prostate cancer screening outcomes.

Chromosomal differences affect how muscle cells respond to testosterone.

Understanding how androgens and their receptors work can lead to improved treatments for skin diseases.

Androgens increase muscle mass by promoting myoblast growth through ornithine decarboxylase.

The conclusion suggests that prostate cancer should be classified by castration status and that new therapies targeting androgen receptor signaling show promise.

Testosterone significantly affects sexual desire in both men and women, but its impact on women is more complex and influenced by psychological factors.

The conclusion is that adult males with muscle weakness and hormonal imbalances should be tested for Kennedy's disease.

Effective treatments for spinal and bulbar muscular atrophy are not yet available; more research is needed.

The gene SDR5A1 is found in scalp hair of both hirsute and normal individuals, but it does not explain differences in hair growth.