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Researchers successfully grew horse skin cells that produce pigment from hair follicle samples.

Enzymes are classified into six types and are essential for many biological processes, with only a few targeted by drugs.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

People with hidradenitis suppurativa often have lower vitamin D levels, weaker hip bones, and altered bone metabolism markers.

VDUP1 is found in skin and hair follicles, interacts with sciellin, and may help regulate skin cell differentiation.

Vitamin D deficiency is common in people with certain types of hair loss, like alopecia areata and female pattern hair loss.

Some lesser-known causes of PCOS include autoimmune issues, genetic mutations, and changes in the body's microbiome.

Localized heat or specific injections can prevent chemotherapy-induced hair loss without affecting cancer treatment.

Vitamin D3 applied to mouse skin caused more wrinkles and sagging due to changes in the skin's outer layer.

No FDA-approved treatments for chemotherapy-induced hair loss existed in 2006; more research was needed.

Increasing ABC transporters in hair follicles may prevent chemotherapy-induced hair loss.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

The research showed that Vitamin D and its receptor are important for healthy bones and normal hair and skin in rats.

A Chinese family had a child with a specific gene mutation causing vitamin D-resistant rickets, but the child improved with calcium and low-dose calcitriol.

Many women with hair loss also have thyroid issues, high blood pressure, and low Vitamin D.

Vitamin D receptor binds similarly to natural and synthetic ligands, affecting gene regulation.

A child with a rare type of rickets showed some improvement with high doses of vitamin D, but such conditions often respond poorly to treatment.

The study found no link between vitamin D deficiency and psoriasis but suggests more research is needed.

Topical vitamin D is effective in treating vitiligo with few side effects.

Rubbing calcipotriol on the skin works better than taking vitamin D pills for treating mild to moderate alopecia areata.

Patients with myotonic dystrophy often have skin problems that suggest early aging and vitamin D issues, and the severity of these problems is linked to their genetic condition and vitamin D levels.

Researchers found that certain lipids, especially vitamin D3, are lower in prematurely grey hair than in pigmented hair.

Removing vitamin D and calcium receptors in mice skin cells slows down skin wound healing.

The extract reduced sebum production and promoted hair growth.

Special particles from umbilical cord stem cells help heal skin wounds in diabetic mice by preventing certain immune cell death.

Low vitamin D receptor levels found in hair loss patients; topical vitamin D treatment suggested.

Possible new treatments for common hair loss include drugs, stem cells, and improved transplants.

Many potential alopecia treatments need more testing to confirm they promote acceptable hair growth with minimal side effects.

Skin cells can help create early hair-like structures in lab cultures.

Genes linked to fibrosis are more active in people with central centrifugal cicatricial alopecia.