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The document explains hair and nail biology, common hair loss conditions and treatments, oral and genital skin diseases, and the risks and treatments associated with squamous cell carcinoma.

Ectodysplasin signaling is crucial for skin appendage development, requiring specific doses and durations.

Hair thinning causes stem cell loss through a process involving Piezo1, calcium, and TNF-α.

Rare ULBP3 gene changes may raise the risk of Alopecia areata, a certain FAS gene deletion could cause a dysfunctional protein in an immune disorder, and having one copy of a specific genetic deletion is okay, but two copies cause sickle cell disease.

Mutations in the WNT10A gene can cause skin, hair, teeth, and other disorders, and may also affect other areas like kidney and cancer, with potential for targeted treatments.

Dlx3 is essential for hair growth and regeneration.

New gene mutations linked to skin conditions were found, bacteria and chemicals may worsen acne, a dog mutation could exist in humans, virus-like elements might be involved in psoriasis, and a vitamin D3 treatment doesn't prevent chemotherapy-related hair loss.

P-cadherin is important for hair growth and health, and its problems can cause hair and skin disorders.

A WNT10A gene mutation leads to ectodermal dysplasia by disrupting cell growth and differentiation.

FGF13 gene changes cause excessive hair growth in a rare condition.

A new genetic area linked to a rare hair loss condition was found on chromosome 13 in a Chinese family.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

RANK-RANKL signaling is essential for hair growth and skin health.

Scientists have found specific genes linked to different hair loss conditions, which could lead to new treatments.

A new gene variant in the DSP gene is linked to a unique type of hair loss.

Some patients with mycosis fungoides or Sézary syndrome experience hair loss, which may be similar to alopecia areata or linked to skin lesions, possibly due to abnormal T cells, and bexarotene can help treat it.

Uncombable hair syndrome causes frizzy hair and can affect the nervous system, eyes, and ears, often co-occurring with other hair, skin, nail, and teeth conditions, and is linked to three specific gene mutations.

EGF and KGF signalling prevent hair follicle formation and promote skin cell development in mice.

Neurohormones help control skin health and could treat skin disorders.

Researchers found 15 new genetic links to skin traits in Japanese women.

CD2 might be a new treatment target for patchy alopecia areata.

Skin problems can be caused or worsened by physical forces and pressure on the skin.

Genetic variations affecting skin structure play a key role in severe acne.

Some congenital hair disorders improve in childhood or with treatments like minoxidil and retinoids, while others like Netherton syndrome and trichothiodystrophy have a poor prognosis.

Bald thigh syndrome in sighthounds is caused by structural defects in hair shafts due to downregulated genes and proteins.

An 8-year-old girl had nail and hair issues that improved without specific treatment, but fungal nail infection was hard to cure.

Skin's epithelial stem cells are crucial for repair and maintenance, and understanding them could improve treatments for skin problems.

Some hair loss disorders are caused by genetic mutations affecting hair growth.

The document concludes that parathyroid diseases have a range of clinical features and outcomes, with some conditions being treatable and others having a high risk of mortality.

Skin patterns form through molecular signals and genetic factors, affecting healing and dermatology.