Search

everythinglearnresearchcommunity

for

Search:↓

Vitamin D boosts a specific gene activity in kidney cells that could improve heart and kidney function.

Researchers found key regions in the mouse hairless gene that control its activity in skin and brain cells, affecting hair follicle function.

Vitamin D receptor binds similarly to natural and synthetic ligands, affecting gene regulation.

Vitamin D and its receptor may help prevent skin cancer.

Vitamin D receptor is important for regulating hair growth and wound healing in mice.

Vitamin D receptor may help protect against UV-induced skin cancer.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

The vitamin D receptor helps maintain hair and bone health even without binding vitamin D.

Lithocholic acid helps hair growth and regeneration in alopecia by activating vitamin D receptors.

Removing part of the vitamin D receptor stops vitamin D from working properly.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

Mice without the vitamin D receptor have bone issues and other health problems, suggesting vitamin D is important for preventing various diseases in humans.

The vitamin D receptor is crucial for bone health and affects various body systems, with mutations potentially leading to disease.

Skin cells produce and activate vitamin D, which regulates skin functions and supports hair growth.

Vitamin D has potential benefits for cancer prevention, heart health, diabetes, obesity, muscle function, skin health, and immune function, but clinical results are mixed and more research is needed.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

LEF1 interacts with Vitamin D Receptor, affecting hair follicle regeneration and this could be linked to hair loss conditions.

The review found no clear link between vitamin D receptor gene variations and polycystic ovary syndrome.

Topical vitamin D is useful for some skin conditions but not effective for others, and more research is needed.

Vitamin D boosts a specific protein in skin cells linked to hair follicles.

The R343H mutation in the VDR gene causes vitamin D-resistant rickets with alopecia by impairing specific gene activity.

Topical vitamin D is effective in treating vitiligo with few side effects.

Vitamin D levels do not significantly relate to the causes of PCOS or DOR in infertile patients.

Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

Vitamin D receptor helps control hair growth and could be used to treat certain skin tumors.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

The study found that certain mutations in the vitamin D receptor can cause rickets and potentially affect hair growth.

Gene therapy helped rats with a specific type of rickets grow hair without severe inflammation.

Vitamin D receptor knockout mice have significant motor impairments but no cognitive deficits.

The vitamin D receptor can work without its usual activating molecule.