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Vitamin D is important for regulating calcium, phosphate, and hair health.

Baldness does not affect vitamin D levels in men.

Patients with myotonic dystrophy often have skin problems that suggest early aging and vitamin D issues, and the severity of these problems is linked to their genetic condition and vitamin D levels.

Vitamin D and calcium are essential for normal hair growth.

Vitamin D boosts a specific protein in skin cells linked to hair follicles.

The vitamin D receptor helps regulate skin and hair health independently of its usual vitamin D ligand.

A genetic mutation caused severe rickets and alopecia in an Indian patient, but high-dose calcium and phosphate treatment improved their condition.

VDUP1 is found in skin and hair follicles, interacts with sciellin, and may help regulate skin cell differentiation.

High Nesfatin-1 and low Vitamin D may increase blood pressure and heart rate in women with PCOS.

Vitamin D boosts a specific gene activity in kidney cells that could improve heart and kidney function.

Vitamin D deficiency is common in women with PCOS and linked to some metabolic problems, but not the main cause of their metabolic issues.

Lack of Vitamin D receptor changes skin structure and increases certain immune cells in the skin.

Vitamin D and calcium are important for quick and effective skin wound healing.

Low vitamin D levels are linked to various types of hair loss, but more research is needed before using it as a treatment.

Vitamin D and calcium are important for skin stem cell function and wound healing.

Vitamin D is important for skin health, but more research is needed to understand its full effects and treatment potential.

People with certain types of hair loss, especially lichen planopilaris and telogen effluvium, as well as African Americans, Asians, and men, are more likely to have severe vitamin D deficiency.

Vitamin D and estrogen may help protect heart and kidney health, and maintaining sufficient vitamin D levels could be especially beneficial for African Americans, postmenopausal women, and people with chronic kidney disease.

Some people have genetic mutations that make them resistant to vitamin D, leading to rickets even with enough vitamin D intake.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

Vitamin D receptor helps control hair growth genes in skin cells.

Vitamin D receptor gene variations are not linked to alopecia areata.

Low vitamin D levels are common in people with Alopecia Areata.

A vitamin D receptor mutation causes rickets and affects immune responses.

Vitamin D could be important for many skin functions and synthetic versions may treat various skin diseases.

A 4-year-old boy with a rare type of rickets and hair loss improved in bone health but not hair growth after vitamin D and calcium treatment.

Low iron levels are a significant risk factor for hair loss, while high vitamin D levels might be a response to hair loss, not a cause.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

Certain variations in the vitamin D receptor gene are linked to an increased risk of polycystic ovary syndrome.

Vitamin D receptor helps protect skin cells from UV damage and supports their growth.