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Research 31–60 of 504
- Skin abnormalities generated by temporally controlled RXRα mutations in mouse epidermis
- Hereditary 1,25-dihydroxyvitamin D-resistant rickets with alopecia in four Egyptian families: report of three novel mutations in the vitamin D receptor gene
- Phenotypic Heterogeneity in 5 Japanese Patients with an Identical Point Mutation in the Vitamin D Receptor Gene
- CRISPR/Cas9-mediated<i>VDR</i>knockout plays an essential role in the growth of dermal papilla cells through enhanced relative genes
- Deletion of vitamin D receptor with calcium sensing receptor in keratinocytes promotes epidermal tumorigenesis by limiting dna repair and oxidative stress response genes
- Vitamin D receptor-mediated control of Soggy, Wise, and Hairless gene expression in keratinocytes
- Physical and Functional Interaction between the Vitamin D Receptor and Hairless Corepressor, Two Proteins Required for Hair Cycling
- Development and progression of alopecia in the vitamin D receptor null mouse
- Hairless Suppresses Vitamin D Receptor Transactivation in Human Keratinocytes
- Vitamin D‐dependent rickets type I and type II
- Hereditary 1,25‐Dihydroxyvitamin D–Resistant Rickets Due to an Opal Mutation Causing Premature Termination of the Vitamin D Receptor
- Interactions of the Vitamin D Receptor with the Corepressor Hairless
- A Humanized Mouse Model of Hereditary 1,25-Dihydroxyvitamin D–Resistant Rickets Without Alopecia
- Presence of a Deletion Mutation (c.716delA) in the Ligand Binding Domain of the Vitamin D Receptor in an Indian Patient with Vitamin D-Dependent Rickets Type II
- Tooth Development Associated with Mutations in Hereditary Vitamin D–Resistant Rickets
- Novel Vitamin D Receptor Mutations in Hereditary Vitamin D Resistant Rickets in Chinese
- Ligand‐independent Regulation of the hairless Promoter by Vitamin D Receptor<sup>†</sup>
- Hereditary vitamin D-resistant rickets in Lebanese patients: the p.R391S and p.H397P variants have different phenotypes
- Effect of calcium supplementation on bone deformity and histopathological findings of skin papules in a pediatric patient with vitamin D–dependent rickets type 2A: a case report
- 6874 A Rare Case Of Hereditary 1,25 (OH)2D Resistant Rickets
- C‐reactive protein as a novel biomarker for vitamin D deficiency in alopecia areata
- Alopecia with Vitamin D-Dependent Rickets Type 2 A: A Case Report
- The role of vitamin D receptor signaling in hair follicle health and alopecia: Current understanding and therapeutic implications
- Cloning of a Functional Vitamin D Receptor from the Lamprey (Petromyzon marinus), an Ancient Vertebrate Lacking a Calcified Skeleton and Teeth
- Hairless protein of Jumonji family and hair loss
- Hereditary 1,25-Dihydroxyvitamin D Resistant Rickets due to a Mutation Causing Multiple Defects in Vitamin D Receptor Function
- The vitamin D receptor functions as a transcription regulator in the absence of 1,25-dihydroxyvitamin D3
- Lack of association between Vitamin D receptor FokI polymorphism and alopecia areata.
- Mechanism of JmjC-containing protein Hairless in the regulation of vitamin D receptor function
- The Nuclear Vitamin D Receptor: Biological and Molecular Regulatory Properties Revealed