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Genetic defects in testosterone production can cause hormonal and developmental disorders, and more research is needed to understand androgen regulation and develop safer treatments.

Carbamates may help treat androgen-dependent conditions by changing how certain lipid enzymes are produced.

Testosterone, progesterone, and levonorgestrel change enzyme levels related to fat production in hamster skin, which could affect skin oil and acne.

Testosterone and dihydrotestosterone treatments can help with penile growth in males with 5α-reductase type 2 deficiency, with dihydrotestosterone being more effective in infancy.

Male hormones can decrease the ability of immune cells to fight bacteria.

Spironolactone treatment increases the amount of testosterone available in the body.

Finasteride and CPA-EE2 equally reduce hirsutism, but affect hormone levels differently.

Both treatments reduced androgen levels and hair growth in women with hirsutism.

A new mutation in the Hr gene causes hair loss in mice, similar to a human hair disorder.

Transdermal testosterone can improve sexual desire in postmenopausal women but lacks long-term safety data and is not FDA-approved for this use.

Testosterone helps human pancreatic cells increase insulin release.

Human mitochondrial DNA polymerase makes very few errors, crucial for preventing degenerative diseases.

The document suggests that targeting the hormone DHT could be a more effective treatment for prostate cancer than targeting testosterone.

New compounds may soon be tested to treat excessive hair growth in women.

Most patients improved with oral contraceptives, but some needed additional treatment.

Obesity changes androgen levels in women with PCOS, leading to higher testosterone relative to androstenedione.

Dihydrotestosterone increases growth and spread of human brain cancer cells, and blocking its formation might help treat this cancer.

Temporal hair loss relates to overall scalp hair loss in women.

5α-Reductase inhibitors do not consistently increase testosterone levels in the blood.

Finasteride reduces prostate size and serum levels, helping manage benign prostatic hyperplasia, without affecting prostate cancer detection. Regular monitoring is needed for users.

New testosterone analogs show promise for male contraception with better activity and potentially fewer side effects.

Many Korean women with female-pattern hair loss have reduced hair density at the temples.

A new gene mutation causes female pseudohermaphroditism due to glucocorticoid resistance.

Finasteride, CPA, and flutamide are all equally effective in reducing excessive hair growth in women.

New pregnane derivatives are effective at inhibiting an enzyme linked to hair loss and reducing oil gland activity.

Different halogens on progesterone derivatives can either block or mimic male hormone effects, depending on their type and amount.

TR3 is mainly found in hair follicle stem cells and may be involved in hair loss.

Certain gene variations in the Vitamin D Receptor are linked to higher risk of female hair loss.

Blocking CRF1 receptors improved male hormone levels and reduced testicular tumor size in men with a specific adrenal condition.

Male pattern baldness is mainly caused by a hormone called dihydrotestosterone (DHT), and drugs that can block this hormone might help prevent hair loss.