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Researchers fixed gene mutations causing a skin disease in stem cells, which then improved skin grafts in mice.

Lgr5 is a marker for active, long-lasting stem cells in mouse hair follicles.

MicroRNA-214 is important for skin and hair growth because it affects the Wnt pathway.

Mutant mice help researchers understand hair growth and related genetic factors.

Disrupting Acvr1b in mice causes severe hair loss and thicker skin.

Genetic mutations can cause hair growth disorders by affecting key genes and signaling pathways.

Raspberry ketone may help grow hair and improve skin elasticity.

SCF and c-Kit decrease in AGA hair follicles, possibly affecting hair pigmentation and growth.

The protein SLC1A3 is important for activating skin stem cells and is necessary for normal hair and skin growth in mice.

PTHrP and its receptor can control blood vessel growth and hair development in mouse skin.

Mice without certain skin proteins had abnormal skin and hair development.

Wnt3a activates certain genes in hair follicle cells, including a newly discovered one, EP2, which may affect hair growth.

The document concludes that understanding how adult stem and progenitor cells move is crucial for tissue repair and developing cell therapies.

DHT stops hair regrowth in mice, similar to human hair loss.

Parathyroid hormones are important for hair growth, but their use in treating hair loss from chemotherapy is still uncertain.

Putting α-methylspermidine on mouse skin can start hair growth.

The document concludes that the immune-inhibitory environment of the hair follicle may prevent melanoma development.

Mice with too much sPLA₂-IIA have hair loss and poor wound healing due to abnormal hair growth and stem cell depletion.

Both main and alternative Wnt signaling are important for regrowing rodent whisker follicles.

Activin B helps start and grow hair follicles in mice.

Scientists can mimic hair disorders by altering genes in lab-grown human hair follicles, but these follicles lack some features of natural ones.

Pigs without the Hairless gene showed skin and thymus changes, useful for studying human hair disorders.

New treatments for vitiligo may focus on protecting melanocyte stem cells from stress and targeting specific pathways involved in the condition.

Certain proteins help nerve cells branch, and other findings relate to cancer, stem cell behavior, and cell division.

Scientists created a new cell line from Cashmere goat hair and found that cytokeratin 13 is a unique marker for certain skin cells.

Transplanting a mix of specific skin cells can significantly improve the repair of damaged hair follicles.

DNA methylation is essential for skin and hair follicle development, and could be a target for treating skin diseases.

Melanocytes produce melanin; their defects cause vitiligo and hair graying, with treatments available for vitiligo.

Hair follicle stem cells can turn into many cell types and may help repair nerve damage and have other medical uses.

The document concludes that alopecia has significant social and psychological effects, leading to a market for hair loss treatments.