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Overexpressing thrombospondin-1 in mice skin prevents UVB-induced skin damage.

Epithelial stem cells are crucial for tissue renewal and repair, and understanding them could improve treatments for damage and cancer.

A protein from certain immune cells is key for new hair growth after skin injury in mice.

Vitamin D receptor is crucial for starting hair growth after birth.

The document concludes that mouse models are crucial for studying hair biology and that all mutant mice may have hair growth abnormalities that require detailed analysis to identify.

Hair growth is controlled by specific gene clusters and proteins, and cysteine affects hair gene expression in sheep.

Blocking a specific protein signal can make hair grow on mouse nipples.

Changes in keratin affect skin health and can lead to skin disorders like blistering diseases and psoriasis.

Nestin identifies specific progenitor cells in hair follicles that can become outer root sheath cells.

Wnt and Shh signaling are key in noggin-induced tumors, and blocking them can slow tumor growth.

The study found that mouse sweat glands develop before birth, mature after birth, and have specific keratin patterns.

Scientists developed tools to observe hair regeneration in real time and assess skin health, using glowing mice and light-controlled genes.

Noggin overexpression delays eyelid opening by affecting cell death and skin cell development.

Blocking BMP signaling causes hair loss and disrupts hair growth cycles.

HPV16 oncogenes disrupt the normal activity of hair follicle stem cells.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Understanding gene expression in hair follicles can reveal insights into hair growth and disorders.

Too much Smad7 changes skin and hair development by breaking down a protein called β-catenin, leading to more oil glands and fewer hair follicles.

Adam10 enzyme is crucial for healthy skin and proper Notch signaling.

HPV genes in mice improve ear tissue healing by speeding up skin growth and repair.

The protein CTCF is essential for skin development, maintaining hair follicles, and preventing inflammation.

KGF and activin are crucial for skin healing and repair.

The document concludes that adult mammalian skin contains multiple stem cell populations with specific markers, important for understanding skin regeneration and related conditions.

RANK-RANKL signaling is essential for hair growth and skin health.

Two-photon microscopy effectively tracks live stem cell activity in mouse skin with minimal harm and clear images.

HPV genes and estradiol increase a cancer-related signaling pathway, which may be targeted for cervical cancer treatment.

The document concludes that recent research has improved understanding of skin diseases and the balance between cell growth and differentiation in the epidermis.

Antizyme reduces tumor growth and normalizes skin cell development affected by MEK.

Activating TERT in mice skin boosts hair growth by waking up hair follicle stem cells.