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Air pollution (PM10) increases skin inflammation and aging by reducing collagen and may trigger a repair response in skin cells.

Acne is caused by genetics, diet, hormones, and bacteria, with treatments not yet curative.

Oestrogens help maintain healthy skin, heal wounds, and may protect against skin aging and cancer.

Tissue-resident memory T cells can protect against infections and cancer but may also contribute to autoimmune diseases.

Estrogens and SERMs can help with skin aging, but their safety and effectiveness need more research.

Different scaffold patterns improve wound healing and immune response in mouse skin, with aligned patterns being particularly effective.

Melanocyte development from neural crest cells is complex and influenced by many factors, and better understanding could help treat skin disorders.

Certain immune system proteins are important for skin healing but can cause problems if there are too many of them.

Toll-like receptors are important for wound healing, but can slow it down in diabetic wounds.

Kennedy's disease leads to muscle weakness and sensory issues, has no cure but manageable symptoms, and future treatments look promising.

Anti-acne medications may work by reducing the activity of a protein involved in acne development.

ADT, especially enzalutamide, may increase the risk of heart rhythm problems and sudden death in men.

Blocking the CXCR3 receptor reduces T cell accumulation in the skin and prevents hair loss in mice.

New findings explain how genetic changes, body clocks, and certain molecules affect tissue response to stress hormones.

Karl E. Weick suggested focusing on everyday events and smaller groups to improve organizational theory and urged the inclusion of nonobvious aspects for better explanations.

There is no clear recommendation for using selenium in cancer patients; it may be beneficial to correct low selenium levels before treatment.

Lymphocytes, especially CD8+ T cells, play a key role in causing alopecia areata, and targeting them may lead to new treatments.

Immunosuppressive and anti-TNF therapies in IBD patients can increase the risk of skin cancer and cause various skin issues.

RANK-RANKL signaling is essential for hair growth and skin health.

High doses of cyproterone acetate increase the risk of brain tumors in women, but the risk decreases after stopping the medication.

Both mouse and rat models are effective for testing alopecia areata treatments.

The article explains the genetic causes and symptoms of various hair disorders and highlights the need for more research to find treatments.

Smad-4 and Smad-7 are key in hair follicle development, with other Smads being less important.

Researchers found 15 new genetic links to skin traits in Japanese women.

Blocking BACE1 and BACE2 enzymes causes hair color loss in mice.

The document concludes that the skin's immune system is complex, involving interactions with hair follicles, nerves, and microbes, and can protect or cause disease, offering targets for new treatments.

Researchers found 71 genetic regions linked to male pattern baldness, which account for 38% of its genetic risk.

Improving the environment and cell interactions is key for creating human hair in the lab.

Brain hormones significantly affect hair color and could potentially be used to prevent or reverse grey hair.

Steroid sex hormones activate matriptase in prostate cancer cells but not in breast cancer cells.