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COVID-19 may harm male fertility and damage the reproductive system.

SARS-CoV-2, the virus causing COVID-19, can affect all endocrine organs and systems, altering their function and potentially leading to disorders. Factors like diabetes and obesity increase infection risk and severity. Understanding these effects is key for effective treatment.

The ACE1 gene variant doesn't affect long-COVID symptoms.

People with diabetes or obesity should manage their conditions carefully as they have a higher risk of severe COVID-19.

Wait 3 months after COVID-19 before trying assisted reproduction and further research is needed on COVID-19's effects on male hormones and fertility.

Traditional Chinese medicinal foods may help manage long-term post-COVID symptoms.

People with lower levels of free testosterone tend to have worse COVID-19 outcomes.

Androgen deprivation therapy doesn't lower the risk of death from COVID-19 in prostate cancer patients.

Women are more likely to have long-term post-COVID symptoms than men.

Men and women get COVID-19 at similar rates, but men tend to get sicker and have a higher risk of dying, while women usually have stronger immune responses and vaccine reactions.

Male hormones affect COVID-19 severity and certain drugs targeting these hormones could help reduce the risk.

NF-κB signaling is crucial in many diseases and can be targeted for new treatments.

People with androgenetic alopecia may have a higher risk of severe COVID-19.

Sex hormones may affect COVID-19 severity and outcomes.

The drug enzalutamide may reduce the ability of the virus causing COVID-19 to enter lung cells.

Mutations in certain receptors can cause diseases and offer new treatment options.

Blocking the virus's entry into cells by targeting certain pathways could lead to early COVID-19 treatments.

Sex hormones may influence COVID-19 severity, with males at higher risk, and certain hormone therapies could potentially treat the virus.

The conclusion is that certain genetic factors and blood types may affect COVID-19 severity, but changes in ACE2 and TMPRSS2 genes are not clearly linked to it.

Male hormones like testosterone may make COVID-19 worse, and testing for sensitivity to these hormones could help predict how severe a patient's symptoms might be. Treatments that reduce these hormones are being explored.

Women with conditions like PCOS may have a higher risk of COVID-19, but treatments like isotretinoin could help those with acne.

Androgens may worsen COVID-19 and hair loss could indicate the disease's severity.

Skin problems in COVID-19 patients are rare and may be due to the body's complex immune response or blood clotting issues.

The conclusion is that individual differences in COVID-19 severity are influenced by factors like age, sex, race, and genetics, which are important for personalized medicine.

The conclusion suggests that prostate cancer should be classified by castration status and that new therapies targeting androgen receptor signaling show promise.

Existing drug dexamethasone may lower death risk in severe COVID-19 cases; more research needed for other drugs.

Betacoronaviruses, like COVID-19, may cause hormone system dysfunction and affect disease susceptibility and severity.

Blocking enzymes that help the virus enter cells could be a promising way to treat COVID-19.

Men are more likely to have severe respiratory viral infections like COVID-19 due to hormonal and genetic differences, while women generally have stronger immune responses.

Men have worse COVID-19 outcomes than women due to genetic and hormonal differences.