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Thymic transplantation normalized some T-cells but not others, maintaining immune function.

The FOXN1 gene is crucial for developing immune cells and preventing immune disorders.

Thymus transplantation successfully restored immune function in infants with FOXN1 deficiency.

Transplant success has improved with better immunosuppressive drugs and donor matching.

Two siblings had a rare immune disorder caused by a FOXN1 gene mutation.

FOXN1 gene mutations cause a rare, severe immune disease treatable with cell or tissue transplants.

FOXN1 gene variants cause low T cells and immune issues from birth.

New treatments for immune disorders caused by FOXN1 deficiency are promising.

A boy with severe immune deficiency and Epstein-Barr virus died from high-grade B-cell lymphoma.

Biotin treatment can significantly improve hair loss and motor functions in certain metabolic disorders.

A 10-month-old boy with a rare combination of genetic conditions has severe immune deficiency and treatment challenges.

Th22 cells are essential for Tβ15-induced hair growth in mice.

Mice with human fetal thymic tissue and stem cells developed symptoms similar to chronic graft-versus-host disease.

Scientists turned rat thymus cells into stem cells that can help repair skin and hair.

A five-month-old boy with Omenn syndrome successfully recovered after a stem cell transplant with reduced intensity conditioning.

Thymic mesenchymal cells have unique gene expression that supports their specific functions in the thymus.

Some patients developed hair loss after islet cell transplant possibly due to their immune-suppressing medications.

T-cell reconstitution after thymus transplantation can cause hair whitening and loss.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

Environmental cues can change the fate and function of epithelial cells, with potential for cell therapy.

Deleting the p63 gene in certain cells causes problems in thymus development and severe hair loss in mice.

New findings suggest targeting IL-23 could treat psoriasis, skin cells can adapt to new roles, direct conversion of skin cells to blood cells may aid cell therapy, removing certain tumor cells could boost cancer immunotherapy, and melanoma may have many tumorigenic cells, not just cancer stem cells.

New cell-based therapies may improve hair loss treatments in the future.

Skin abnormalities can indicate immunodeficiency due to shared origins with the immune system.

Adult esophageal cells can start to become like skin cells, with a key pathway influencing this change.

Increasing sebum production might help reduce fat and improve metabolism.

Lgr5 is a marker for active, long-lasting stem cells in mouse hair follicles.

Deleting Snai2 and Snai3 causes fatal autoimmunity.

Hair follicles help attract immune cells to minor skin injuries.

Skin issues can indicate immune system problems.