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Non-immune dermal cells dominate, epidermal cells increase after day 9, and certain immune cells persist beyond inflammation in wound-induced hair follicle regeneration.

Alopecia Areata is an autoimmune condition causing hair loss with no cure and treatments that often don't work well.

CRAC channels are crucial for the development and function of specialized immune cells, preventing severe inflammation and autoimmune diseases.

Lower CD200R1 on certain immune cells is linked to more severe rheumatoid arthritis and immune imbalance.

New treatments for hair loss from alopecia areata may include targeting immune cells, using stem cells, balancing gut bacteria, applying fatty acids, and using JAK inhibitors.

Immune cells called Treg cells are essential for hair growth and regeneration.

Immune cells are crucial for hair growth and preventing hair loss.

Linalool in personal care products may worsen frontal fibrosing alopecia by damaging hair follicle stem cells and triggering harmful immune responses.

Researchers found a specific T cell receptor linked to severe drug reactions like Stevens-Johnson syndrome when patients take carbamazepine.

Stem cell offspring help control their parent stem cells, affecting tissue health, healing, and cancer.

Special immune cells called Tregs can help prevent lung scarring by blocking a specific growth factor.

Increasing Treg cells in the skin does not cure hair loss from alopecia areata in mice.

Deleting the p63 gene in certain cells causes problems in thymus development and severe hair loss in mice.

Specific immune cells cause alopecia areata and blocking certain proteins can prevent it.

Increasing the levels of a protein called FGF9 can promote hair growth, but humans may not respond the same way due to a lack of certain cells.

Linalool in personal care products may contribute to hair loss by damaging hair follicle stem cells and triggering harmful immune responses.

ILC1-like cells may contribute to hair loss in alopecia areata.

ILC1-like cells can independently cause alopecia areata by affecting hair follicles.

ILC1-like cells may contribute to hair loss in alopecia areata and could be new treatment targets.

Skin bacteria can help wound healing by activating certain immune cells.

Pemphigus vulgaris involves specific immune cells and B cells that produce antibodies causing skin blisters.

Small molecule DMF improves psoriasis and multiple sclerosis, adult skin cells can be made to grow new hair, certain skin cells initiate hair growth, IL-17C controls gut health and can cause skin inflammation, and skin cells produce IL-17 that can lead to psoriasis.

Loss of a specific protein in skin cells causes symptoms similar to psoriasis.

Early treatment with the right dose of stem cells can reduce lupus symptoms.

ILC1-like cells can cause alopecia areata by disrupting hair follicle immunity, suggesting a new treatment approach.

Mice treatments didn't grow hair, a patient treatment may affect immune response, and people with hair loss often feel anxious or depressed.

New treatments for alopecia areata may target specific immune cells and pathways involved in hair loss.

Tofacitinib helped most patients with alopecia areata regrow hair and changes in immune cells were linked to the treatment's effectiveness.

EGCG may help treat alopecia areata by blocking certain immune responses and reducing specific harmful immune cells.

Some cells may slow melanoma growth, a protein could affect skin pigmentation, a gene-silencing method might treat hair defects, skin bacteria changes likely result from eczema, and a defensin protein could help treat multiple sclerosis.