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Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

Flutamide caused damage to male rat reproductive organs and may affect fertility.

Mice without certain skin enzymes have faster hair growth and bigger eye glands.

Higher SHBG levels are linked to less severe hair loss in women, but vitamin D levels don't seem to affect hair loss.

Finasteride is more harmful to male reproductive health than minoxidil.

Obesity changes androgen levels in women with PCOS, leading to higher testosterone relative to androstenedione.

Male androgenetic alopecia involves hair follicle miniaturization due to DHT, with potential treatments using inhibitors and blockers.

SAHA syndrome is a condition in women involving skin and hair issues, often related to hormonal imbalances, and is treated based on the underlying cause.

Most hirsutism cases are due to PCOS, and treatment focuses on lowering testosterone and blocking its effects.

The document concludes that managing female hyperandrogenism requires a combination of identifying the cause, lifestyle changes, medication, and cosmetic treatments.

Treating sleep apnea in young obese women with PCOS can improve heart health and insulin sensitivity.

Neurosteroids show promise for treating epilepsy and more research is needed.

PCOS is a complex condition linked to hormonal imbalance and insulin resistance, with weight loss being important for management.

Some herbs show promise as antiandrogenic agents.

Skin problems in PCOS, like excess hair, acne, and hair loss, may not always indicate high male hormone levels and need careful diagnosis for proper treatment.

Finasteride affects offspring's antioxidant enzymes in epididymis, possibly disrupting sperm maturation.

New treatments for PCOS show promise, but no current medication treats all aspects of the condition.

Finasteride reduces alcohol withdrawal severity in male mice but increases it in female mice.

PFOS exposure disrupts hormone levels and enzyme activities in juvenile frogs, affecting males and females differently.

Finasteride may help reduce impulsivity caused by pramipexole.

New nonsteroidal molecules can potentially increase dihydrotestosterone in neurons by blocking certain enzymes, without affecting prostate and seminal vesicle weight.

Prostate cancer can progress even with low testosterone due to internal hormone production in the tumor.

Certain pesticides and fungicides can interfere with male hormone functions, potentially causing reproductive issues in male rats.

Finasteride may help reduce opioid use.

Increasing TSPO in the brain reduces anxiety and depression.

Metformin should be used for PCOS mainly in those with glucose intolerance, and has limited benefits for infertility or hirsutism.

Glioblastoma cells can make androgens, which might help the tumor grow.

AMH levels can't reliably tell the difference between LOCAH and all types of PCOS in women with excessive hair growth.

The modified Ferriman-Gallwey method is a useful tool for diagnosing hirsutism.

High-dose dutasteride reduces PMDD symptoms by stabilizing neurosteroid levels.