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Dutasteride mesotherapy helps treat male hair loss but may harm sperm and sexual function.

PDRN, derived from salmon sperm, shows promise in healing wounds, reducing inflammation, and regenerating tissues, but more research is needed to understand its mechanisms and improve its use.

Finasteride treatment in male rats can reduce fertility and affect sperm development in their offspring.

Men who went through puberty later had lower sperm counts and altered hormone levels, possibly affecting fertility.

Men and women in infertile couples feel using donor sperm may cause more stress and marital issues than using donor eggs.

The combination of cyproterone acetate and testosterone enanthate is highly effective in preventing sperm production and could be a good reversible male contraceptive.

Finasteride changes antioxidant enzyme expression, possibly affecting sperm protection in rats.

Neuronatin is found in various cells of rat tissues and has a unique location in sperm cells.

A man with testotoxicosis was fertile despite low FSH levels, suggesting high testosterone may allow sperm production without FSH.

Gerbils on a fat-deficient diet had altered fatty acid levels, hair loss, reduced sperm, and lower body weight.

The April 2014 issue of "Fertility and Sterility" discussed various reproductive health topics, including hormone therapy benefits, sperm and genetic factors in male infertility, and the link between PCOS and diabetes.

Dihydrotestosterone and 5α-Reductase play a role in hair loss and prostate health, and finasteride can increase hair growth in men without affecting sperm production, but it doesn't work for postmenopausal women with hair loss.

Finasteride, at doses of 5mg or higher, may negatively affect male fertility by reducing the expression of certain genes involved in sperm production.

Long-term testosterone therapy can cause hormone suppression, affect prostate and heart health, and alter physical characteristics, but does not increase prostate cancer risk and needs more research for full risk assessment.

A subtle androgen receptor abnormality can allow normal male development and sometimes fertility despite partial androgen resistance.

COVID-19 might affect male fertility, but more research is needed to understand the full impact.

Testosterone replacement therapy can prevent men from fathering children and should not be used by those wanting to stay fertile.

Finasteride may cause male infertility, stopping it can help.

Wait 3 months after COVID-19 before trying assisted reproduction and further research is needed on COVID-19's effects on male hormones and fertility.

Some skin medications can harm male fertility, but they don't seem to cause birth defects from father's exposure.

Finasteride exposure harms tadpole reproduction and hormone balance.

The document concludes that hormonal biomarkers are key for diagnosing hyperandrogenemia in women and hypogonadism in men.

Dihydrotestosterone treatment can help penis growth in boys with 5α-reductase deficiency but doesn't fully normalize size after puberty.

Testosterone increases acidification in the vas deferens by up-regulating V-ATPase.

Testosterone is vital for male health, and its deficiency is linked to aging and common health issues.

Melatonin improved sheep reproduction, being more beneficial and cost-effective in males.

Finasteride does not negatively affect male reproductive function.

5α-reductase-2 deficiency causes ambiguous genitalia at birth and affects male sexual development, but individuals often develop male characteristics at puberty.

Stem cells regenerate tissues and their behavior varies by environment, suggesting the hematopoietic system model may need revision.

A man's fertility improved after he stopped taking finasteride but worsened again when he restarted the medication.