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Foxi3 expression in developing teeth and hair is controlled by the ectodysplasin pathway.

TCDD disrupts skin stem cells, causing skin issues like chloracne.

NK cells play a role in skin diseases like eczema and psoriasis.

Lack of Vitamin D receptor changes skin structure and increases certain immune cells in the skin.

HR protein causes abnormal hair cycles by increasing Tgf-β2 and reducing miR-31.

Overexpressing Merkel cell virus proteins in human hair follicles can create clusters of cells that resemble Merkel cell cancer.

Hair can regrow in large wounds through a process similar to how hair forms in embryos, and understanding this could lead to new treatments for hair loss or scarring.

Understanding genetic mutations helps diagnose and treat skin disorders like ichthyosis.

The method increases stem-like cells for better skin regeneration.

Using blood-based implants improves skin healing and reduces scarring.

Scientists found a new type of skin cell that could help with skin repair and these cells work better with a certain protein.

Uncombable hair syndrome causes frizzy hair and can affect the nervous system, eyes, and ears, often co-occurring with other hair, skin, nail, and teeth conditions, and is linked to three specific gene mutations.

Nonpalmoplantar skin cells can be made to express keratin 9 by interacting with palmoplantar fibroblasts.

The workshop highlighted the genetic links and psychological impacts of hair loss and skin disorders.

Ichthyoses are genetic skin disorders that affect the skin's barrier function.

Targeting TGFβ can improve skin immunity in older people.

Low oxygen levels improve the function of hair and skin cells when they are in direct contact.

Magnesium oxide-infused membranes help heal wounds faster by reducing inflammation and promoting skin and hair follicle growth.

A new EDA gene mutation was found in a Chinese family with a specific skin disorder.

Scientists created a long-lasting stem cell line from human hair that can turn into different skin and hair cell types.

The harlequin ichthyosis mouse mutation causes thick skin and early death, resembling a human skin disorder.

A litter of cats had a hair condition similar to a mouse mutation, leading to hair loss and abnormal hair and skin.

Early over-expression of FoxN1 harms immune and skin development.

Sox2-positive dermal papilla cells have unique characteristics and contribute more to skin and hair follicle formation than Sox2-negative cells.

CYP2B12 enzyme in skin cells converts arachidonic acid into specific bioactive lipids.

A gene mutation in mice causes permanent hair loss and skin issues.

A new mutation in the PLEC gene causes a rare condition with skin blistering, muscle weakness, and hair loss.

GLI1 might protect against the start of skin cancer and is not linked to cancer severity.

Foxn1 is important for fat development, metabolism, and wound healing in skin.

p120-catenin helps control skin inflammation by regulating cadherin levels.