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Scientists have developed a new method to detect steroid abuse in athletes using cell-based tests, which could be the future of anti-doping methods.

Lichen planopilaris and frontal fibrosing alopecia are likely the same disease with different clinical appearances.

Regenerative pharmacology, which combines drugs with regenerative medicine, shows promise for repairing damaged body parts and needs more interdisciplinary research.

Traction may not be the only cause of cicatricial marginal alopecia.

The curved nonrotary punch is better for hair extraction in people with tightly curled hair.

The Foxn1(-/-) nude mouse shows disrupted and expanded skin stem cell areas due to high Lhx2 levels.

Notch signalling helps skin cells differentiate and prevents tumors.

Vitamin D receptor helps control hair growth genes in skin cells.

Removing a specific gene in certain skin cells causes hair loss on the body by disrupting normal hair development.

Mice without the vitamin D receptor have bone issues and other health problems, suggesting vitamin D is important for preventing various diseases in humans.

The study concluded that hair growth in mice is regulated by a stable interaction between skin cell types, and disrupting this can cause hair loss.

Harlequin mutant mice have hair loss due to low AIF protein levels and retroviral element activity.

The vitamin D receptor helps regulate skin and hair health independently of its usual vitamin D ligand.

The vitamin D receptor can work without its usual activating molecule.

Eating a high-fat fish oil diet caused mice to lose hair due to a specific immune cell activity in the skin linked to a protein called E-FABP.

Mice without certain skin enzymes have faster hair growth and bigger eye glands.

Mice with human skin protein K8 had more skin problems and cancer.

Vitamin D receptor is crucial for starting hair growth after birth.

Lack of cystatin M/E causes thin hair and dry skin.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

Hairless protein is crucial for healthy skin and hair, and its malfunction can cause hair loss.

Tight junctions are key for skin protection and controlling what gets absorbed or passes through the skin.

Damaged hair follicles make mice more prone to skin inflammation and skin cancer after UV exposure.

TNF family proteins are crucial for the development of skin features like hair, teeth, and mammary glands.

Keratin protein production in cells is controlled by a complex system that changes with cell type, health, and conditions like injury or cancer.

Deleting the PTEN gene in mice causes nerve cells to grow larger and heal better after injury, but may cause overgrowth and hair loss in older mice.

Mutations in Plcd1 and Plcd3 together cause severe hair loss in mice.

Vitamin D receptor interacts with certain dietary components to help prevent diseases and regulate hair growth.

Mice lacking the PPARγ gene in their fat cells had almost no fat tissue, severe metabolic problems, and abnormal development of other fat-related tissues.

The Foxn1 gene mutation causes hairlessness and immune system issues, and understanding it could lead to hair growth disorder treatments.