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The nude gene causes skin cell overgrowth and improper development, leading to hair and urinary issues.

AIRE protein, defective in APECED patients, is found in skin and hair cells and interacts with cytokeratin 17.

Notch1 signaling is impaired in hidradenitis suppurativa, affecting skin and hair cells.

Coats' Plus is a genetic disorder with eye abnormalities, brain calcification, poor growth, bone and skin issues, and movement disorders.

CD18-deficient mice developed psoriasis-like skin disease, useful for studying inflammatory skin disorders.

Artificial dermis used for hair transplantation can reconstruct scalp defects effectively without the need for tissue expansion.

New RIPK4 gene mutations were found to cause a type of skin and limb birth defect.

Two enzyme defects in biotin metabolism cause severe skin, hair, and metabolic issues.

Technique effectively treats deformities, achieves re-pigmentation, and releases scar contractures.

FoxN1 gene is crucial for proper thymus structure and normal skin appearance.

Antiyeast treatments work better for seborrheic dermatitis than steroids, and other findings include increased skin bacteria under plastic wrap, a nasal cell defect in Staphylococcus aureus carriers, quick resolution of certain skin reactions, high rubber latex allergy in spina bifida patients, and glyceryl monopentadecanoate's effectiveness for male hair loss.

FoxN1 gene is essential for proper thymus structure and preventing hair loss.

Local flaps are effective for covering small to large scalp and forehead defects with good cosmetic results.

Using skin grafts from behind the ear for oral surgery in two patients with jaw injuries led to successful healing and good results.

The chapter concludes that certain skin diseases in bovines cause symmetrical, non-painful lesions and temporary hair loss due to stress.

The superficial temporal artery flap is a reliable method for reconstructing complex facial defects with minimal complications.

A mutation in mice causes hair loss and skin issues due to a defect in a gene affecting cell adhesion.

Patients with this syndrome can have different responses and worsening resistance to treatment over time.

C/EBPalpha and C/EBPbeta are crucial for normal skin and oil gland cell development in adult mice.

CD98hc's role in skin health decreases with age.

The model better predicts how water-loving and fat-loving substances move through the skin by including tiny pores and hair follicle paths.

The skin condition Ulerythema ophryogenes did not improve with treatment in a patient with other birth defects.

Some skin medications can harm male fertility, but they don't seem to cause birth defects from father's exposure.

A patient with Rothmund-Thomson syndrome showed skin changes, hair loss, and dental defects but no cataracts or skeletal issues.

Some cells may slow melanoma growth, a protein could affect skin pigmentation, a gene-silencing method might treat hair defects, skin bacteria changes likely result from eczema, and a defensin protein could help treat multiple sclerosis.

Fat cells help recruit healing cells and build skin structure during wound healing.

PDGFA/AKT signaling is important for the growth and maintenance of certain skin fat cells.

The document concludes that adult mammalian skin contains multiple stem cell populations with specific markers, important for understanding skin regeneration and related conditions.

Human skin and hair color variation is mainly due to melanin produced by a few genes, with melanin protecting against sun damage.

Hair follicles are essential for skin health, aiding in hair growth, wound healing, and immune function.