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Leflunomide is more likely to help treat alopecia areata than cause it.

Understanding the immune-related causes of Alopecia Areata has led to potential treatments like JAK inhibitors.

The document concludes that pig iPSCs show promise for transplant therapies and the field is advancing in controlling cell behavior for biology and medicine.

Cell death shapes skin stem cell environments, affecting inflammation, repair, and cancer.

Skin spheroids with both outer and inner layers are key for regrowing skin patterns and hair.

Hair aging is caused by stress, hormones, inflammation, and DNA damage affecting hair growth and color.

Dexamethasone affects hair growth by altering levels of proteins that either promote or inhibit hair follicle growth.

Substances from fat-derived stem cells can promote hair growth and counteract hormone-related hair loss by activating a key hair growth pathway.

The document concludes that the development of certain tumors is influenced by genetic background and that a specific gene modification can lead to tumor regression and reduced growth.

Alopecia areata is an autoimmune condition causing hair loss, linked to genetic factors and immune system issues, with no cure yet.

Skin cell behavior is influenced by the tightness of nearby cells, affecting their growth and development.

The document concludes that alopecia has significant social and psychological effects, leading to a market for hair loss treatments.

Blocking the CCR5 receptor may be a new way to treat hair loss from alopecia areata.

Blocking Oncostatin M's role in the JAK-STAT pathway can stimulate hair growth in mice.

Blocking Prostaglandin D₂ (PGD₂) could help treat hair loss.

The document explains how shampoos clean, rinses condition, one-step shampoos offer convenience, and hair growth promoters aim to prevent hair loss, emphasizing the importance of scientific evidence for their effectiveness.

Estrogen Receptor ß (ERß) is the main hormone controller in human skin and hair follicles, not Estrogen Receptor α (ERα) or the Androgen Receptor (AR).

Animal models have helped understand hair loss from alopecia areata and find new treatments.

Lichen Planopilaris and Frontal Fibrosing Alopecia may help us understand hair follicle stem cell disorders and suggest new treatments.

Hair loss in androgenetic alopecia is caused by genetic factors and androgen excess, and can be treated with combined therapies.

Aging mice have slower hair regeneration due to changes in signal balance, but the environment, not stem cell loss, controls this, suggesting treatments could focus on environmental factors.

Both vitiligo and alopecia areata involve an immune response triggered by stress and specific genes, with treatments targeting this pathway showing potential.

Blocking the CXCR3 receptor reduces T cell accumulation in the skin and prevents hair loss in mice.

Exosomes are crucial for protecting sensory hair cells in the inner ear.

The study explains how minoxidil sulfate causes vasodilation in rabbits by opening potassium channels and inhibiting calcium channels.

Gray hair may be caused by lower antioxidant activity in hair cells.

Dieckol from Ecklonia cava may help hair growth and could be a potential hair loss treatment.

A new hair loss treatment uses tiny needles to deliver a drug-loaded lipid carrier, promoting hair growth more effectively than current treatments.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

Quercitrin may help treat hair loss by promoting hair growth and improving cell health.