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Deleting the PTEN gene in mice causes nerve cells to grow larger and heal better after injury, but may cause overgrowth and hair loss in older mice.

Trichilemmal carcinoma shares genetic changes with other skin cancers, suggesting similar causes and potential treatments.

Trichilemmal carcinoma shares genetic traits with other skin cancers, suggesting similar treatment options.

Trichilemmal carcinoma shares genetic traits with other skin cancers, suggesting similar treatment options.

Inactive hair follicle stem cells help prevent skin cancer.

Exosomes help nerve fibers grow by affecting specific cell signaling pathways.

PTEN helps control the number and health of skin stem cells by working with the protein BMAL1.

Understanding where cancer cells come from helps create better prevention and treatment methods.

Fibroblast growth factor receptor signaling controls cell development and repair, and its malfunction can cause disorders and cancer, but it also offers potential for targeted therapies.

The drug enzalutamide may reduce the ability of the virus causing COVID-19 to enter lung cells.

Tumor suppressors help control inflammation in cancer and restoring their function could lead to new treatments.

Removing Sprouty genes in mice causes various hormone-related issues but does not increase cancer risk by one year of age.

Low oxygen areas help maintain and protect blood stem cells by using a simple sugar breakdown process for energy and managing their activity levels.

Melanoma's complexity requires personalized treatments due to key genetic mutations and tumor-initiating cells.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive stomach cancer.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive forestomach cancer.

WWP2 is crucial for tooth development in mice.

The document concludes that the immune-inhibitory environment of the hair follicle may prevent melanoma development.

Macrophages help regrow hair by activating stem cells using AKT/β-catenin and TNF.

New treatments for advanced skin cancer are improving patient outcomes, but drug resistance and finding the right treatment combinations are still big challenges.

HDAC6 helps keep ovarian follicles dormant, extending female fertility.

Deleting the MAD2L1 gene in mice led to rapid tumor growth despite chromosomal instability.

TGFβ's manipulation of inflammation and immune cells affects cancer spread, suggesting new treatment strategies and biomarkers.

Removing vitamin D and calcium receptors in mice skin cells slows down skin wound healing.

Deleting the MAD2L1 gene is tolerated in certain mouse cancer models.

The document concludes that stem cell inactivity is actively controlled and important for tissue repair and balance.

Young women in West Bengal, India, with PCOS often have estrogen resistance, leptin receptor issues, folate deficiency, T2DM, and acanthosis, commonly linked to obesity.

PDGF signaling is crucial for maintaining and renewing hair follicle stem cells, which could help treat hair loss.

A mutation in the Adam10 gene causes freckle-like spots on Hairless mice.

The document concludes that understanding genetic mutations in the PI3K-AKT-mTOR pathway can lead to better diagnosis and treatment for certain genetic skin disorders.