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FERONIA and LRX proteins help control cell growth in plants by regulating vacuole expansion.

Inactive stem cells in hair follicles and muscles can avoid detection by the immune system.

Two rare genetic diseases cause severe rickets in children due to defects in vitamin D metabolism.

Hair follicles are normally protected from the immune system, but when this protection fails, it can cause hair loss in alopecia areata.

Frizzled receptors are essential for various body development processes and maintaining certain body functions.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

Progeroid mouse models show signs of early aging similar to humans, helping us understand aging better.

Hairless protein can block vitamin D activation in skin cells.

Three genes in Arabidopsis are important for plant growth and development by affecting sugar attachment to proteins.

HOXC13 is essential for hair and nail development by regulating Foxn1.

Loss of a specific protein in skin cells causes symptoms similar to psoriasis.

Folliculin deficiency causes problems with cell division and positioning due to disrupted RhoA signaling and interaction with p0071.

GmMAX3b gene in soybeans boosts nodulation and affects hormone levels.

Keratin proteins are essential for keeping the cells in the human colon healthy and stable.

A specific enzyme is crucial for the bean plant's relationship with certain beneficial soil bacteria and fungi.

Gab1 protein is crucial for hair growth and stem cell renewal, and Mapk signaling helps maintain these processes.

Studying premature aging syndromes helps understand human aging and suggests potential treatments.

Prostasin has two roles in skin: one for normal skin development without needing activation, and another for proper hair growth that requires activation.

The conclusion is that genetic testing is important for diagnosing and treating various genetic hair disorders.

A specific gene mutation causes congenital hair loss.

Vitamin D Receptor is needed for hair growth in mice but not for skin stem cell maintenance.

Black-tailed deer have four types of pelages and molt twice a year.

Horse skin stem cells combined with platelet-rich plasma improve skin healing.

A gene variant increases the risk of a type of hair loss by affecting hair protein production.

A genetic mutation caused severe rickets and alopecia in an Indian patient, but high-dose calcium and phosphate treatment improved their condition.

Activating Nrf2 in skin cells speeds up wound healing by increasing the growth of certain stem cells.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

A new mutation in the ST14 gene broadens the understanding of ichthyosis-hypotrichosis syndrome.

Adding a second method to PROTACs could improve cancer treatment.