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Lindera strychnifolia root extract may help balance scalp bacteria and potentially reduce hair loss.

Both treatments for alopecia areata showed similar modest effectiveness.

Quercetin effectively treated and prevented hair loss in mice.

Human dermal γδT-cells respond to stress in hair follicles, contributing to hair loss.

Japanese patients with alopecia areata often have a higher BMI and consume more vitamin C, fruit, and retinol, which may affect their condition's development or severity.

AIRE deficiency causes hair loss similar to alopecia areata in mice.

Certain daily habits like stress, diet, and sleep can affect the severity of hair loss in alopecia areata.

A protein called sFRP4 can slow down hair regrowth.

A new mouse model for vitiligo helps study immune responses and potential treatments.

New digital tools are improving the diagnosis and understanding of irreversible hair loss conditions.

Men with androgenetic alopecia have different scalp oils and microbes compared to those without.

Alopecia areata involves both innate and adaptive immunity, with specific genes linked to the disease.

Combining LFQS and IPL is more effective and faster for treating melasma than LFQS alone.

Alopecia areata patients have more Propionibacterium acnes and less Staphylococcus epidermidis on their scalps.

Two rare bald spots on the back of the scalp were found to be lupus, not alopecia areata.

PRP treatment may help with alopecia areata, especially in newer cases.

Manipulating cell cleanup processes could help treat hair loss.

No single ideal JAK inhibitor for alopecia areata has been determined; JAK3 inhibitors may be promising with fewer side effects.

CXCL12 protein slows down hair growth through its receptor CXCR4. Blocking this can potentially increase hair growth.

Alopecia areata is an autoimmune disease where T cells attack hair follicles.

Hair follicles prevent NK cell attacks to avoid hair loss.

The MAGE3 hypothesis for alopecia areata did not lead to a significant breakthrough.

Cyclosporin extends hair growth in mice, but high-dose corticosteroids block this effect.

The cause of Frontal fibrosing alopecia, a type of hair loss, is complex, likely involving immune responses and genetics, but is not fully understood.

This type of hair loss is probably often missed and treatments reducing inflammation might work well.

The review suggests that other immune cells besides CD8+ T cells may contribute to alopecia areata and that targeting regulatory cell defects could improve treatment.

Innate lymphoid cells type 1 may contribute to alopecia areata.

Thyroid hormone affects skin health, with too little causing rough, pale skin and too much leading to smooth, thin skin, and may also impact wound healing and skin conditions.

Thyroid hormones affect skin texture, hair and nail growth, and can cause skin diseases related to thyroid problems.

Skin's Regulatory T cells are crucial for maintaining skin health and could be targeted to treat immune-related skin diseases and cancer.