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The model suggests that scalp tension could lead to hair loss, with factors like blood vessel hardening, enlarged oil glands, and poor microcirculation also playing a role. It also hints at a possible link between skull shape and baldness pattern.

Androgens can both increase body hair and cause scalp hair loss.

Androgens can cause hair growth in some areas but hair loss on the scalp.

Thicker hair grows faster; hair loss patients have slower growth.

PHAT may improve hair growth better than PRP alone.

A specific RNA molecule blocks hair growth by affecting a protein related to hair loss conditions.

Early exposure to germs may protect against autoimmune diseases, lack of sex increases alcohol preference in fruit flies, a potential baldness treatment could involve blocking a specific receptor, skin memory cells help prevent re-infection, high-fat diets can affect brain cells related to weight, and the link between social status, stress, and heart disease in primates is unclear.

Estrogen therapy can reduce skin aging but has cancer risks.

Androgenetic alopecia involves genetics, hormones, and can be treated with medications or surgery.

Androgens can both promote and prevent hair growth due to differences in gene expression in hair follicles.

Minoxidil, finasteride, and low-level laser light therapy are effective FDA-approved treatments for hair loss.

Minoxidil and finasteride effectively treat hair loss.

Oxidative stress damages hair and contributes to aging, and managing it can help maintain hair health.

Male hormones, particularly DHT, are linked to male pattern hair loss, and treatments like finasteride can help, but they don't work for postmenopausal women's hair loss, which may have different causes.

Male pattern hair loss caused by follicular miniaturization; early diagnosis and treatment can reduce psychological burden.

Beard cells, unlike scalp cells, produce growth factors in response to testosterone, which may explain differences in hair growth.

Estrogens generally inhibit hair growth and improve skin quality, but their exact effects on hair follicles are complex and not fully understood.

Use "female pattern hair loss" term, assess androgen excess, treat with minoxidil and other medications if needed.

Male pattern baldness is an unintended side effect of the body's use of androgens for muscle growth, especially in those genetically prone to it.

Increased PPARGC1α relates to hair thinning in common baldness.

Both surgical and non-surgical hair loss treatments lack a standard treatment plan and sufficient patient satisfaction data.

Certain miRNAs might be involved in a hair loss condition called frontal fibrosing alopecia and could possibly help in its diagnosis.

Prostaglandin D2, found in higher levels in bald scalps, stops hair growth and could be a target for treating hair loss.

Gypenosides from Gynostemma pentaphyllum were found to have anti-aging effects, increasing skin collagen and reducing wrinkles.

Higher IGF-1 levels in hair follicles link to better finasteride results for hair loss.

Baldness caused by male hormones in female-to-male transsexuals doesn't increase the risk of heart disease.

The paper suggests that hair loss might be caused by skull growth, not just DHT's effect on hair follicles, and calls for more research.

IVIG therapy was linked to hair regrowth in two patients with hair loss, but it's not recommended due to its risks and costs.

Androgens can both increase and decrease hair growth in different parts of the body.

Estrogen Receptor ß (ERß) is the main hormone controller in human skin and hair follicles, not Estrogen Receptor α (ERα) or the Androgen Receptor (AR).