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Mild exercise reduces pain in older rats through a brain chemical, while intense exercise reduces pain in all rats through a different pain-blocking process.

Mitochondrial problems in diabetic nerve damage might cause pain by lowering the production of certain nerve-related steroids.

Benzothiazepines like diltiazem reduce anxiety in mice by making neurosteroids.

Diabetic neuropathy in mice is linked to poor mitochondria function and lower brain hormone production.

Neurosteroids can influence behavior by modulating brain inhibition, with potential for treating psychiatric disorders.

DHEA improved brain function and behavior in old monkeys and had additional health benefits.

Lower levels of certain brain chemicals are linked to worse PTSD symptoms in men.

Researchers created a new method to measure brain steroids, finding higher levels of certain steroids and changes due to a drug.

Finasteride almost fully depletes allopregnanolone in rat brains and enhances 20α-DHP, but doesn't change 3α-DHP levels.

The availability of certain hormones and specific stimulation patterns affect long-term synaptic changes in the male rat brain.

Seeing trauma causes fear in mice by lowering their natural fear-reducing hormones.

Finasteride and dutasteride reduce neurosteroid production, possibly helping treat glioblastoma.

Finasteride use may lead to less alcohol consumption in men with lasting sexual side effects.

Medications for enlarged prostate can cause sexual side effects like reduced libido, erectile dysfunction, and ejaculatory problems.

A brain-produced steroid causes increased scratching in mice with a skin condition similar to eczema.

Pregnancy and nursing increase certain brain activities in rats, but these changes disappear when the babies are taken away.

Agmatine may help reduce seizures linked to hormone changes in female rats.

Neurosteroids help regulate oxytocin levels, especially during stress and pregnancy, to protect against premature labor.

Gene expression differences in PFS patients suggest a potential organic cause for symptoms.

Ferulic acid can reduce seizures and depression-like behaviors in catamenial epilepsy by balancing hormones and brain enzyme activity.

Increasing TSPO in the brain reduces anxiety and depression.

Neurosteroids play a key role in controlling the brain-adrenal gland activity in pregnant sheep, both in normal and stressful situations.

Sex hormones affect brain cells differently in males and females.

Short-term finasteride use in male rats caused anxiety, depression, and memory problems.

Trilostane may help delay epilepsy development by increasing certain brain chemicals.

Finasteride reduces alcohol withdrawal severity and anxiety in mice, but may increase withdrawal severity in some cases.

New compounds were made that block an enzyme linked to breast cancer better than existing treatments.

Certain steroids in the brain affect mood and symptoms of depression, and treatments targeting these steroids show promise for improving these symptoms.

Neuroactive steroids show promise for treating mental and neurological disorders by targeting GABA_A receptors.

Activating TSPO helps protect the retina from damage caused by high eye pressure in glaucoma.