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Deleting the p63 gene in certain cells causes problems in thymus development and severe hair loss in mice.

Hair follicle development and microbes help regulatory T cells gather in newborn skin.

Thymic transplantation normalized some T-cells but not others, maintaining immune function.

FoxN1 overexpression in young mice harms immune cell and skin development.

Foxn1 gene regulation is crucial for thymus development but not for hair growth.

CRAC channels are crucial for the development and function of specialized immune cells, preventing severe inflammation and autoimmune diseases.

Thymic mesenchymal cells have unique gene expression that supports their specific functions in the thymus.

Human prenatal skin develops an immune network early on that helps with skin formation and healing without scarring.

FOXN1 gene mutations cause a rare, severe immune disease treatable with cell or tissue transplants.

Foxp3 is crucial for regulatory T cell function, and targeting these cells may help treat immune disorders.

miR-22, a type of microRNA, controls hair growth and its overproduction can cause hair loss, while its absence can speed up hair growth.

T cells and bacteria in the gut and skin help maintain health and protect against disease.

MAIT cells may help fight COVID-19 but also contribute to severe inflammation.

Your skin is like an ecosystem, with its own community of microbes and substances that interact and affect its health.

Too much Smad7 can cause serious changes in skin tissues, including problems with hair growth, thymus shrinkage, and eye development issues.

Skin abnormalities can indicate immunodeficiency due to shared origins with the immune system.

The nude gene causes skin cell overgrowth and improper development, leading to hair and urinary issues.

Understanding how melanocyte stem cells work could lead to new treatments for hair graying and skin pigmentation disorders.

Newborn screening and gene therapy are expected to improve outcomes for Omenn syndrome patients.

Damaging mutations in NFKB2 cause a severe and distinct form of primary immunodeficiency with early-onset and often ACTH-deficiency.

Changes to the Foxp3 protein affect how well regulatory T cells can control the immune system, which could help treat immune diseases and cancer.

RANKL causes lymph nodes to grow by making certain cells multiply.

Men generally have more severe COVID-19 cases and higher death rates than women due to biological differences.

Loss of a specific protein in skin cells causes symptoms similar to psoriasis.

FOXN1 mutations cause severe immunodeficiency, hair loss, nail issues, and thymus defects.

Two siblings had a rare immune disorder caused by a FOXN1 gene mutation.

Too much Smad7 changes skin and hair development by breaking down a protein called β-catenin, leading to more oil glands and fewer hair follicles.

The skin protects the body and is constantly renewed by stem cells; disruptions can lead to cancer.

Pannexin 3 helps skin and hair growth by controlling a protein called Epiprofin.

Mutations in the hairless gene in mice affect its expression and lead to a range of developmental issues in multiple tissues.