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The nude gene causes skin cell overgrowth and improper development, leading to hair and urinary issues.

Most hair follicle stem cells do not protect their DNA by dividing it unevenly.

The document suggests treating individuals with nonclassic congenital adrenal hyperplasia who show symptoms, especially those related to excess male hormones.

Foxi3 expression in developing teeth and hair is controlled by the ectodysplasin pathway.

Gene therapy has potential as a future treatment for Hutchinson-Gilford progeria syndrome.

Oral minoxidil shows promise in treating monilethrix-related hair loss.

Cells from a skin condition can create new hair follicles and similar growths in mice, and a specific treatment can reduce these effects.

The research concluded that selection significantly shaped the genetic variation of the X chromosome, with certain regions affected by past selective events.

Ca_v1.2 affects hair growth and could be a target for hair loss treatments.

Rapamycin treatment helps with mitochondrial disease by reducing PKC levels.

Blocking the FGF5 gene in sheep leads to more fine wool and active hair follicles due to changes in certain cell signaling pathways.

Mouse skin color ranges from pink to black, depending on their hair growth cycle.

Researchers found a new mutation causing total hair loss from birth.

Hair follicle regeneration may slow tumor growth.

P-cadherin is important for hair growth and health, and its problems can cause hair and skin disorders.

No link found between aromatase gene and female hair loss.

Changing the diet of mice lacking the enzyme CBS can affect symptoms related to the genetic condition.

Haplogroup X found in Altaian population supports Amerindian origin.

A defective protein in progeria causes cell death and atherosclerosis, but a treatment targeting cell stress may reduce these effects.

Genetic mutation and carcinogen treatment are both needed for skin cancer to develop in these specific mice.

A new mutation in the hairless gene causes hair loss and skin wrinkling in mice.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

Modified pep7, named EPM peptide, effectively promotes hair growth at low concentrations and works well with minoxidil.

Mutation in hairless gene may increase hair loss risk.

A specific thyroid hormone resistance mutation may be linked to different types of hair loss.

The best animal model for studying male-pattern baldness is the stumptailed macaque, not rats or mice.

A Chinese family had a child with a specific gene mutation causing vitamin D-resistant rickets, but the child improved with calcium and low-dose calcitriol.

Chemical exposure may contribute to the rise in atopic diseases and needs more research.

Progranulin overexpression leads to shorter, thinner hair and increased cell death in mouse hair follicles.

The R343H mutation in the VDR gene causes vitamin D-resistant rickets with alopecia by impairing specific gene activity.