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Mice with a Gsdma3 gene mutation have thicker skin and longer hair follicle openings due to increased β-catenin levels.

Mutations in Plcd1 and Plcd3 together cause severe hair loss in mice.

A litter of cats had a hair condition similar to a mouse mutation, leading to hair loss and abnormal hair and skin.

Mutations in iRhom2 affect hair and skin in mice and are linked to esophageal cancer, with ADAM17 playing a crucial role.

Blocking IL-17 can reduce skin inflammation in a mouse model of pityriasis rubra pilaris.

The L457(3.43)R mutation in the human lutropin receptor causes increased activity and hormone insensitivity, leading to precocious puberty.

Mutant CDP/Cux protein causes hair defects and reduced male fertility in mice.

The androgen receptor gene doesn't affect women with recurrent spontaneous abortions, but having a mutant genotype might protect against it.

Mutant cells in hair follicles are influenced by their location and interactions with surrounding cells.

Some mutant mice have hair with abnormal cross-linking, mainly in the cuticle, not affecting other hair parts.

The Arabidopsis rhd2 mutant is more sensitive to drought because of changes in cell membrane proteins and cell structure.

Targeted siRNA therapy may be a promising treatment for KID syndrome by reducing mutant gene expression and improving cell communication.

The research found that Atypical Progeroid Syndrome has unique symptoms and is not caused by the buildup of a certain mutant protein.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

Hair grows faster in the morning and is more vulnerable to damage from radiation due to the internal clock in hair follicle cells.

Photodynamic therapy is the preferred treatment for skin precancer due to its effectiveness and safety.

A new mouse model for vitiligo helps study immune responses and potential treatments.

UV photography can help identify people at higher risk for skin cancer, and male pattern baldness at age 45 is linked to a higher risk of certain skin cancers.

Activating Wnt in skin cells controls the number of hair follicles by directing cell movement and fate.

Mutations in keratin genes cause skin disorders, but new treatments show promise.

Androgenetic alopecia is linked to a higher risk of coronary heart disease due to certain genetic factors and high homocysteine levels.

Skin can heal wounds without hair follicle stem cells, but it takes a bit longer.

Phospholipase A2 is necessary for the correct placement of PIN proteins in plant roots, affecting root growth.

Circadian clock genes are important for hair growth and may affect aging-related hair loss and graying.

Hair follicle regeneration may slow tumor growth.

The scraggly mutation causes hair loss and skin defects in mice.

SGK3 is essential for proper hair growth and health.

The SHJH hr mice with a mutated Hr gene show signs of faster skin aging due to poor antioxidative protection.

Certain changes in the SHBG gene may increase the risk of PCOS in Iraqi women.

The balance between cell renewal and differentiation controls the growth of cancerous cells in mouse skin.