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Capsaicin, found in chili peppers, can slow down hair growth by affecting skin cells and hair follicles.

Skin-derived precursors in hair follicles come from different origins but function similarly.

Androgen receptor activity blocks Wnt/β-catenin signaling, affecting hair growth and skin cell balance.

Scientists found cells in hair that are key for growth and color.

The protein CCN2 controls hair growth by affecting hair follicle formation and stem cell activity in mice.

Skin lesions in Carney complex are likely caused by a specific group of skin cells that promote pigment production due to a genetic mutation.

Mast cells play a significant role in hair loss conditions like male pattern hair loss and alopecia areata.

The Peripilar sign does not indicate perifollicular infiltrate in hair loss from Androgenetic Alopecia.

Removing Mediator 1 from certain mouse cells causes teeth to grow hair instead of enamel.

Alopecia areata and vitiligo share immune system dysfunction but differ in specific immune responses and affected areas.

Fire Needle Therapy may help bring back skin color in vitiligo by affecting cell growth signals.

The study identified unique genes in hair follicle cells and their environment, suggesting these genes help organize cells for hair growth.

Chemotherapy damages hair follicles, causing hair loss and other cellular changes.

Bmpr2 and Acvr2a receptors are crucial for hair retention and color.

Skin lesions in Carney Complex are caused by a gene change in some skin cells that leads to increased pigmentation and may lead to tumors.

The research identifies genes linked to wool quality in sheep and provides insights to improve wool production.

The document concludes that more research is needed to reduce frequent hospital visits, addiction medicine education improves with specific training, early breast cancer surgery findings are emerging, nipple smears are not very accurate, surgery for older melanoma patients doesn't extend life, a genetic condition in infants can often be treated with one drug, doctors are inconsistent with blood clot medication, a certain gene may protect against cell damage, muscle gene overexpression affects many other genes, and some mitochondrial genes are less active in mice with tumors.

EdnrB signaling helps melanocyte stem cells regenerate and could be targeted to treat pigmentation issues.

Wnt signaling is crucial for pigmented hair regeneration by controlling stem cell activation and differentiation.

The Wnt/β-catenin pathway can activate melanocyte stem cells and may help regenerate hair follicles.

The research found ways to activate melanocyte stem cells for potential treatment of skin depigmentation conditions.

Different types of inactive melanocyte stem cells exist with unique characteristics and potential to develop into other cells.

The study found markers indicating that cells responsible for hair color are differentiating in specific areas of the hair follicle.

Beige and leaden pigment genes act within melanocytes, affecting pigment patterns.

Hair grays due to oxidative stress and fewer functioning melanocytes.

Melanoma development can be linked to the breakdown of skin's melanin-producing units.

Gray hair is caused by reduced melanin production or transfer issues, linked to aging and possibly health conditions, with treatments focusing on color camouflage.

Researchers successfully isolated and identified key markers of stem cell-enriched human hair follicle bulge cells.

As people age, their hair follicles produce less pigment, leading to gray and white hair, due to factors like reduced enzyme activity and damage to melanocyte DNA.

Gambogic Amide helps maintain hair color and promotes hair growth.