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A virus protein can activate a pathway that may lead to abnormal hair follicle development.

Certain microRNAs may protect against hair loss in alopecia areata and could be potential treatment targets.

The Hairless gene is crucial for healthy skin and hair growth.

Some wound-healing cells can turn into fat cells around new hair growth in mice.

β-catenin is essential for stem cell activation and proliferation in hair follicles.

Early development of hair, teeth, and glands involves specific signaling pathways and cellular interactions.

MIM is crucial for hair follicle formation and regeneration by controlling cilia formation and hedgehog signaling through its interaction with Cortactin and Src.

TNF family proteins are crucial for the development of skin features like hair, teeth, and mammary glands.

The TGF-β family helps control how cells change and move, affecting skin, hair, and organ development.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

Skin fat has important roles in hair growth, skin repair, immune defense, and aging, and could be targeted for skin and hair treatments.

The document concludes that the skin's immune system is complex, involving interactions with hair follicles, nerves, and microbes, and can protect or cause disease, offering targets for new treatments.

Chemotherapy often causes hair loss, nail changes, and mouth issues, but these are usually manageable.

Disrupting Stat3 in hair follicle stem cells greatly reduces skin tumor formation.

Hormones during pregnancy and lactation keep skin stem cells inactive, preventing hair growth.

Scalp cooling is largely ineffective in preventing hair loss from breast cancer chemotherapy.

Wnt/ß-catenin signaling is crucial for regulating skin stem cells and hair growth, with the right levels and timing needed for proper function.

Hair loss involves immune responses, inflammation, and disrupted signaling pathways.

Researchers created a mouse model of a type of rickets that does not cause hair loss.

The environment around melanocyte stem cells is key for hair regeneration and color, with certain injuries affecting hair color and potential treatments for pigmentation disorders.

Mice with LSS deficiency showed hair loss and cataracts, similar to humans, and can help in understanding and treating this condition.

Different sPLA2 enzymes affect immunity, skin and hair health, reproduction, and may be potential targets for therapy.

The document suggests that certain protein deficiencies and scalp blistering in Epidermolysis Bullosa may cause hair loss.

5α-Reductase inhibitors can help treat hair loss, acne, and prostate issues by reducing DHT levels.

Mutations in Plcd1 and Plcd3 together cause severe hair loss in mice.

Bald thigh syndrome in sighthounds is caused by structural defects in hair shafts due to downregulated genes and proteins.

The study concluded that changing the culture conditions can cause sika deer skin cells to switch from a flat to a 3D pattern, which is important for creating hair follicles.

Hair growth-related cells need the enzyme SCD1 to help maintain the area that supports hair growth.

17β-estradiol may help hair growth by increasing cannabinoid receptor type 1.

Sex hormones affect hair and feather growth and may help manage alopecia and hormone-dependent cancers.