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Melatonin can increase cashmere yield by altering gene expression and restarting the growth cycle early.

African American women have a higher risk of preterm delivery than Caucasian women, and inflammatory stimuli affect gene expression in cells related to PCOS, showing a heightened inflammatory state in women with PCOS.

A blood pressure drug, diltiazem, may also help treat influenza.

Melatonin affects certain genes and pathways involved in cashmere goat hair growth.

The research suggests immune system changes and specific gene expression may contribute to male hair loss, proposing potential new treatments.

Planarian regeneration begins with a specific gene activation caused by injury, essential for healing and tissue regrowth.

The vitamin D receptor is involved in multiple body functions beyond calcium regulation, including immune response and rapid reactions not related to gene activity.

Researchers found four genes that could help diagnose severe alopecia areata early.

A new mutation in the TMEM173 gene and a risk allele in IFIH1 cause a unique set of immune-related symptoms.

Cicatricial alopecia, a permanent hair loss condition, is mainly caused by damage to specific hair follicle stem cells and abnormal immune responses, with gene regulator PPAR-y and lipid metabolism disorders playing significant roles.

Rats can't grow new hair follicles after skin wounds, unlike mice, due to differences in gene expression and response to WNT signaling.

Acne patients' skin, both with and without lesions, shows a strong immune response and higher antimicrobial activity.

UV-B light increases inflammation-related substances in acne-related skin cells.

Human skin cells produce proenkephalin, which changes with environmental factors and skin diseases.

ADM scaffolds help skin heal by promoting a healing-type immune response.

The document concludes that while there are promising methods to control CRISPR/Cas9 gene editing, more research is needed to overcome challenges related to safety and effectiveness for clinical use.

Alopecia areata is likely an autoimmune disease with unclear triggers, involving various immune cells and molecules, and currently has no cure.

RXRα is crucial for proper immune response and links diet to immune function.

Hair follicles are normally protected from the immune system, but when this protection fails, it can cause hair loss in alopecia areata.

Inactive stem cells in hair follicles and muscles can avoid detection by the immune system.

Dandruff is linked to increased T cells and weakened immune protection in hair follicles.

Oxidative stress and immune dysfunction are linked to both Hashimoto's thyroiditis and polycystic ovary syndrome, with diet and specific treatments important for managing these conditions.

Certain immune cells contribute to severe hair loss in chronic alopecia areata, with Th17 cells possibly having a bigger impact than cytotoxic T cells.

Men are more likely to have severe respiratory viral infections like COVID-19 due to hormonal and genetic differences, while women generally have stronger immune responses.

The study suggests that analyzing DNA can help treat hair loss, but more research is needed.

Tofacitinib helped most patients with alopecia areata regrow hair and changes in immune cells were linked to the treatment's effectiveness.

New research has found 14 genes linked to the risk of developing alopecia areata, improving understanding and treatment options.

Vitamin A may influence hair loss conditions like alopecia, but more research is needed to understand how.

Finasteride affects frog testes by increasing testosterone, decreasing 5α-DHT, and impacting genes related to reproduction and other functions.

SCF and ET-1 together significantly increase skin pigmentation and melanin production.