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Alopecia in patients with epidermolysis bullosa varies in severity and is often caused by skin blistering or trauma.

Skin health and repair depend on the signals between skin stem cells and their surrounding cells.

The conclusion is that the nuclear lamina and LINC complex in skin cells respond to mechanical signals, affecting gene expression and cell differentiation, which is important for skin health and can impact skin diseases.

The document concludes that treatments for female hair loss and excess body hair are available, but managing expectations is important.

Hair follicle biology advancements may lead to better hair growth disorder treatments.

Skin stem cells are crucial for maintaining and repairing the skin and hair, using a complex mix of signals to do so.

The document concludes that skin stem cells are important for hair growth and wound healing, and could be used in regenerative medicine.

Stem cell offspring help control their parent stem cells, affecting tissue health, healing, and cancer.

Chitin and chitosan are useful in cosmetics for oral care, haircare, and skincare, including UV protection and strength improvement.

The document concludes that understanding hair follicle biology can lead to better hair loss treatments.

A new microneedle patch made from hair proteins helps regrow hair faster and better than current treatments.

The Sonic hedgehog pathway is crucial for new hair growth during mouse skin healing.

The article concludes that the wool follicle is a valuable model for studying tissue interactions and has potential for genetic improvements in wool production.

MicroRNA-214 is important for skin and hair growth because it affects the Wnt pathway.

Male hormones and their receptors play a key role in hair loss and skin health, with potential new treatments being explored.

The p75 neurotrophin receptor is important for hair follicle regression by controlling cell death.

Mutations in the PADI3 gene are linked to a higher risk of scarring hair loss in women of African descent.

Notch/CSL signaling controls hair follicle differentiation through Wnt5a and FoxN1.

Fetal skin heals without scarring due to unique cells and processes not present in adult skin healing.

EGF and KGF signalling prevent hair follicle formation and promote skin cell development in mice.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

Mice lacking a key DNA methylation enzyme in skin cells have a lower chance of activating stem cells necessary for hair growth, leading to progressive hair loss.

The nude gene causes skin cell overgrowth and improper development, leading to hair and urinary issues.

Melanocyte development from neural crest cells is complex and influenced by many factors, and better understanding could help treat skin disorders.

Dermal exosomes with miR-218-5p boost hair growth by controlling β-catenin signaling.

Skin problems can be caused or worsened by physical forces and pressure on the skin.

The document concludes that mouse models are crucial for studying hair biology and that all mutant mice may have hair growth abnormalities that require detailed analysis to identify.

Hair problems can be caused by genetics or the environment, and treatment should focus on the cause and reducing hair damage.

FA2H is essential for normal fur and sebum production in mice.

Researchers found a new gene, hacl-1, that is active in mouse hair follicles during hair growth and may be important for hair biology.