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A rare skin tumor with bone formation was successfully removed without recurrence.

Loss of TET2 increases the risk of skin and oral cancer.

Researchers discovered potential origins and new treatments for skin cancer, including biomarkers for melanoma and therapies that reduce tumor growth.

New findings suggest targeting IL-23 could treat psoriasis, skin cells can adapt to new roles, direct conversion of skin cells to blood cells may aid cell therapy, removing certain tumor cells could boost cancer immunotherapy, and melanoma may have many tumorigenic cells, not just cancer stem cells.

The document concludes that understanding nail anatomy is key for diagnosing nail diseases, early signs of nail melanoma may allow for less aggressive treatment, and specific genetic mutations are important in thyroid cancer prognosis and treatment.

Dimethylarsinic acid speeds up skin tumor growth in certain mice.

The extracellular matrix affects where tumors can start in the body.

The document concluded that stem cells are crucial for skin repair, regeneration, and may help in developing advanced skin substitutes.

Markers CRABP1, Nestin, and Ephrin B2 are present in skin cancer environments and may influence their development.

Higher fluence in hair removal damages hair follicles more, while lower fluence mimics natural hair regression, with long-term IPL treatments effectively reducing hair.

Mobile phone radiation may cause DNA damage in human ear hair follicle cells.

Dermal papilla cells are key for hair growth and could help us understand and treat hair loss.

Implanting hair-follicle stem cells in mice brains helped repair brain bleeding and reduced brain inflammation.

Hair follicle stem cells have significant potential for treating various disorders.

Blocking both main energy pathways can stop hair follicle stem cell-induced skin cancer growth.

The protein aPKCλ is crucial for keeping hair follicle stem cells inactive and for hair growth and regeneration.

Hair follicles can regrow in wounded adult mouse skin using a process like embryo development.

Hair follicles are complex, dynamic mini-organs that help us understand cell growth, death, migration, and differentiation, as well as tissue regeneration and tumor biology.

Hair follicle development is controlled by interactions between skin tissues and specific molecular signals.

Nerve signals are crucial for hair follicle stem cells to become skin stem cells and help in wound healing.

Hair and nail cells share similar proteins, indicating a common differentiation pathway.

The conclusion is that certain cell interactions and signals are crucial for hair growth and regeneration.

Epithelial-mesenchymal interactions control hair growth cycles through specific molecular signals.

A protein from certain immune cells is key for new hair growth after skin injury in mice.

Noggin is necessary to start the hair growth phase in skin after birth.

Bone Morphogenetic Proteins (BMPs) help control skin health, hair growth, and color, and could potentially be used to treat skin and hair disorders.

Wnt signaling controls whether hair follicle stem cells stay inactive or regenerate hair.

Wnt ligands are crucial for hair growth and repair.

Hair follicle stem cells are key for hair and skin regeneration, can be reprogrammed, and have potential therapeutic uses, but also carry a risk of cancer.

CDP is crucial for lung and hair follicle cell development.