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A substance called VIP might protect hair follicles from being attacked by the immune system, and problems with VIP signaling could lead to hair loss in alopecia areata.

ALRN-6924 may prevent hair loss caused by chemotherapy.

A defective gene causes hair loss and taste insensitivity in BTBR mice.

A 6-year-old girl was diagnosed with a rare hair disorder called monilethrix.

The meeting discussed vitamin D3's role in fighting tuberculosis, potential treatments for skin conditions like psoriasis, and hair follicle regeneration as a possible solution for hair loss.

Mouse skin can produce and process serotonin, with variations depending on hair cycle, body location, and mouse strain.

Mice with too much Claudin-6 have skin barrier problems and abnormal hair growth.

An enzyme other than TNAP might be responsible for vitamin B-6 metabolism in the skin.

Mouse stem cells from hair follicles can improve wound healing and reduce scarring.

Wounds can regenerate hair in young mice, but this ability declines with age, offering insights for improving tissue regeneration in the elderly.

AIRE protein, defective in APECED patients, is found in skin and hair cells and interacts with cytokeratin 17.

Topical tofacitinib is effective in promoting hair growth for non-scarring alopecia.

Some medications might reverse gray hair, especially those that reduce inflammation or stimulate pigment production, and vitamin B might help.

Etretinate therapy can cause hair loss by disrupting the hair growth cycle.

Certain mouse strains develop a skin condition similar to a human hair loss disease due to genetic defects.

Hair graying is caused by the loss of pigment cells due to poor maintenance of stem cells in the hair follicle.

Hair color production is closely linked to the active growth phase of hair in mice and may also influence hair growth itself.

Mutant CDP/Cux protein causes hair defects and reduced male fertility in mice.

All-trans retinoic acid causes hair loss by increasing TGF-β2 in hair follicle cells.

Using a special stem cell formula on the scalp once a month for six months helped people with hair loss grow more hair.

Special and immunohistochemical stains are not routinely needed for diagnosing hair disorders.

Over half of the children had abnormal hair under a microscope, with many having genetic hair conditions.

T-cell reconstitution after thymus transplantation can cause hair whitening and loss.

Smoking is linked to more hair loss and premature graying compared to nonsmokers.

Tissue expansion for head and neck reconstruction has good blood supply and doesn't need capsule removal, but expect temporary hair loss with normal growth resuming after 6-8 months.

Cadmium chloride pollution can cause skin disorders, speed up aging, and prevent hair growth.

Vitamin D3 and its receptor are important for hair growth, and understanding receptors could help treat hair graying and skin cancer.

β-catenin in the dermal papilla is crucial for normal hair growth and repair.

The hedgehog signaling pathway is crucial for hair growth but not for the initial creation of hair follicles.

Hair ages due to genetics and environmental factors, leading to graying and thinning, with treatments available for some conditions.