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Thyroid hormones help hair grow, reduce hair loss, and increase hair pigment.

Stress increases nerve fibers and immune cell activity in mouse skin, possibly worsening skin conditions.

Wnt and Notch signaling help wound healing by promoting cell growth and regulating cell differentiation.

Too much Smad7 can cause serious changes in skin tissues, including problems with hair growth, thymus shrinkage, and eye development issues.

Fetal wound healing changes with development, affecting inflammation and collagen, which may influence scarring.

Stress increases a factor in mice that leads to hair loss, and blocking this factor may prevent it.

Overexpressing Dsg3 in mice skin causes excessive cell growth and abnormal skin development.

PDGFA/AKT signaling is important for the growth and maintenance of certain skin fat cells.

Skin-derived precursors in hair follicles come from different origins but function similarly.

A genetic region near the PAX1 gene is linked to a higher risk of scoliosis in females.

MicroRNA-214 is important for skin and hair growth because it affects the Wnt pathway.

Laminin-511 is crucial for early hair growth and maintaining important hair development signals.

One minoxidil-sensitive potassium channel exists in human hair follicles.

Scientists turned mouse stem cells into skin cells that can grow into skin layers and structures.

The study found that two enzymes linked to hair loss are located in different parts of the scalp, supporting a common treatment's effectiveness.

Prolactin affects hair growth and skin conditions, and could be a target for new skin disease treatments.

A genetic change in the EDAR gene causes the unique hair traits found in East Asians.

Androgens can both stimulate and cause hair loss, and understanding their effects is key to treating hair disorders.

Mutant CDP/Cux protein causes hair defects and reduced male fertility in mice.

A WNT10A gene mutation leads to ectodermal dysplasia by disrupting cell growth and differentiation.

The p75 neurotrophin receptor is important for hair follicle regression by controlling cell death.

Notch/CSL signaling controls hair follicle differentiation through Wnt5a and FoxN1.

Oxidative stress affects hair loss in men with androgenetic alopecia.

Fatty acid transport protein 4 is essential for skin and hair development.

All-trans retinoic acid causes hair loss by increasing TGF-β2 in hair follicle cells.

Low-oxygen conditions and ECM degradation products increase the healing abilities of perivascular stem cells.

A new mutation in the human glucocorticoid receptor reduces its function and causes resistance to glucocorticoids.

Prolactin affects hair growth cycles and can cause early hair follicle regression.

Mouflon rams mature gradually with changes in body, horns, and hormones linked to age and seasons, reaching full sexual maturity well after puberty.

Human beard hair medulla contains a unique and complex mix of keratins not found in other human tissues.