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The document concludes that the skin's immune system is complex, involving interactions with hair follicles, nerves, and microbes, and can protect or cause disease, offering targets for new treatments.

Stress may trigger hair loss by affecting immune protection in hair follicles.

The study concluded that immune cells attacking hair follicles cause hair loss in alopecia, with genetics and environment also playing a role, and highlighted the potential of certain treatments.

Alopecia areata causes hair loss due to an immune attack on hair follicles, influenced by genetics and environment.

A specific immune response helps control mite populations on the skin, maintaining healthy hair follicles.

The document concludes that mouse models are helpful but have limitations for skin wound healing research, and suggests using larger animals and genetically modified mice for better human application.

Alopecia areata is likely an autoimmune disease with unclear triggers, involving various immune cells and molecules, and currently has no cure.

The current understanding of frontal fibrosing alopecia involves immune, genetic, hormonal factors, and possibly environmental triggers, but more research is needed for effective treatments.

Alopecia areata causes patchy hair loss and involves immune system disruptions.

Vitamin D3 analogs can promote hair growth in mice genetically prone to hair loss.

Alopecia areata is likely caused by a combination of genetic factors and immune system dysfunction, and may represent different diseases with various causes.

Stress turns hair white by depleting color-giving cells in hair follicles through a specific neurotransmitter related to the body's stress response.

Hair loss in Androgenetic Alopecia is caused by genetics, aging, and lifestyle, leading to hair follicle shrinkage and related health risks.

The Foxn1 gene mutation causes hairlessness and immune system issues, and understanding it could lead to hair growth disorder treatments.

The enzymes Tet1, Tet2, and Tet3 are important for the development of hair follicles and determining hair shape by controlling hair keratin genes.

Skin fat has important roles in hair growth, skin repair, immune defense, and aging, and could be targeted for skin and hair treatments.

Two siblings both had a rare case of alopecia areata at the same time.

Scientists turned mouse stem cells into skin cells that can grow into skin layers and structures.

Two-photon microscopy effectively tracks live stem cell activity in mouse skin with minimal harm and clear images.

EGCG may help treat alopecia areata by blocking certain immune responses and reducing specific harmful immune cells.

Rare changes in the KRT82 gene are linked to a higher risk of Alopecia Areata.

Alopecia areata is an autoimmune disease where T cells attack hair follicles.

The skin's layers protect, sense, and regulate the body's internal balance, but can be prone to cancer.

Alopecia areata is an autoimmune disease causing patchy hair loss, often with other autoimmune disorders, but its exact causes are unknown.

Advanced human skin models improve drug development and could replace animal testing.

Fat cells are important for tissue repair and stem cell support in various body parts.

Skin organoids are a promising new model for studying human skin development and testing treatments.

The skin and thymus develop similarly to protect and support immunity.

Mice genetically modified to produce more CD109 in their skin had less inflammation and better healing with less scarring.

Permanent hair dye may cause total hair loss.