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LHX2 is essential for hair follicle development, controlled by NF-κB and TGFβ2 signaling.

Lipocalin-Type Prostaglandin D2 Synthase (L-PGDS) is a protein that plays many roles in the body, including sleep regulation, pain management, food intake, and protection against harmful substances. It also affects fat metabolism, glucose intolerance, cell maturation, and is involved in various diseases like diabetes, cancer, and arthritis. It can influence sex organ development and embryonic cell differentiation, and its levels can be used as a diagnostic marker for certain conditions.

MOF controls key genes for skin development by regulating mitochondrial and ciliary functions.

A defective gene causes hair loss and taste insensitivity in BTBR mice.

Mutations in the HEPHL1 gene cause abnormal hair and cognitive issues.

The study concludes that the EDA2R gene is activated by p53 during chemotherapy but is not necessary for chemotherapy-induced hair loss.

Mice without the IL-6 gene had more hair growth after injury due to higher activity of a related protein, Stat3.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

The tumor suppressor gene FLCN affects mitochondrial function and energy use in cells.

A special gene region controls the re-emergence of a primitive wool type in Merino sheep, improving their wool yield and adaptability.

Deleting the Hoxc13 gene in frogs shows its crucial role in developing skin structures similar to hair.

Removing the Crif1 gene in mouse skin disrupts skin balance and hair growth.

The vitamin D receptor is crucial for bone health and affects various body systems, with mutations potentially leading to disease.

Cannabinoid receptor-1 signaling is essential for the survival and growth of human hair follicle stem cells.

Hair and skin can regenerate without bulge stem cells due to other compensating cells.

CaBP1 and CaBP2 are important for maintaining hearing by supporting continuous calcium currents and nerve signaling in the ear.

Significant progress was made in understanding PXE, but effective treatments are still needed.

The workshop highlighted the genetic links and psychological impacts of hair loss and skin disorders.

Hair loss and aging are caused by the breakdown of a key protein in hair stem cells.

A mother's PPARγ is crucial for preventing harmful milk that can cause inflammation and growth problems in babies.

miR-22, a type of microRNA, controls hair growth and its overproduction can cause hair loss, while its absence can speed up hair growth.

The vitamin D receptor helps regulate skin and hair health independently of its usual vitamin D ligand.

The document concludes that understanding how hair follicles naturally die and regenerate is important for insights into organ development and could impact health and disease treatment.

The document concludes that stem cells and their environments are crucial for skin and hair health and have potential for medical treatments.

Cells in hair follicles help create fat cells in the skin by releasing a protein called Sonic Hedgehog.

Animal models help study psoriasis but have limitations and don't fully mimic the human disease.

TRPV4 helps cells repair tissue and reduce scarring by controlling calcium levels.

Dicer is crucial for hair growth in mice.

CaBP1 and CaBP2 are important for continuous hearing by preventing inactivation of calcium currents in ear cells, with CaBP2 also able to restore hearing when reintroduced.

Lack of Cisd2 disrupts calcium balance in cells, leading to poorly functioning neutrophils.