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Cannabinoid receptor-1 signaling is essential for the survival and growth of human hair follicle stem cells.

CaBP1 and CaBP2 are important for maintaining hearing by supporting continuous calcium currents and nerve signaling in the ear.

Significant progress was made in understanding PXE, but effective treatments are still needed.

Mutations in the HEPHL1 gene cause abnormal hair and cognitive issues.

The workshop highlighted the genetic links and psychological impacts of hair loss and skin disorders.

Removing the VDR gene in skin cells reduces their growth and affects hair-related genes.

The timing of when the gene Bmal1 is active affects aging and survival, with its absence during development, not adulthood, leading to premature aging.

The study concludes that the EDA2R gene is activated by p53 during chemotherapy but is not necessary for chemotherapy-induced hair loss.

ING5 is crucial for stem cell maintenance and preventing certain cancers.

Mice without the IL-6 gene had more hair growth after injury due to higher activity of a related protein, Stat3.

Blocking the FGF5 gene in sheep leads to more fine wool and active hair follicles due to changes in certain cell signaling pathways.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

The PP2A-B55α protein is essential for brain and skin development in embryos.

The document concludes that mouse models are crucial for studying hair biology and that all mutant mice may have hair growth abnormalities that require detailed analysis to identify.

The research found that a specific skin cell type not only triggers hair growth but also controls hair color, and that aging can lead to hair loss and color changes.

Vitamin A may influence hair loss conditions like alopecia, but more research is needed to understand how.

A mother's PPARγ is crucial for preventing harmful milk that can cause inflammation and growth problems in babies.

Endocannabinoids help control mast cell activity in human skin.

Hair loss and aging are caused by the breakdown of a key protein in hair stem cells.

TET enzymes are important for skin and hair development by controlling gene activity in specific areas.

Scientists restored fertility in male mice lacking a key fertility gene by using a modified gene.

The vitamin D receptor helps regulate skin and hair health independently of its usual vitamin D ligand.

The document concludes that stem cells and their environments are crucial for skin and hair health and have potential for medical treatments.

miR-22, a type of microRNA, controls hair growth and its overproduction can cause hair loss, while its absence can speed up hair growth.

Removing REDD1 in mice increases skin fat by making fat cells larger and more numerous.

The document concludes that understanding how hair follicles naturally die and regenerate is important for insights into organ development and could impact health and disease treatment.

The ITGB6 gene is important for tissue repair and hair growth, and mutations can lead to enamel defects and other health issues.

γδ T cells help with hair growth during wound healing in mice.

TRPV4 helps cells repair tissue and reduce scarring by controlling calcium levels.

Animal models help study psoriasis but have limitations and don't fully mimic the human disease.