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Melatonin and photobiomodulation are effective for treating Alopecia X in German Spitz dogs.

Natural products show promise for new hair loss treatments.

Removing Lrig1-positive stem cells in mice causes temporary loss of sebaceous glands.

Hair cloning for hair loss is not yet a practical solution.

Fibrosis in the bulge area of hair follicles can cause hair thinning in male pattern baldness, and drugs that inhibit fibrosis might help reverse this.

Removing integrin α3β1 from hair stem cells lowers skin tumor growth by affecting CCN2 protein levels.

Mouse hair follicle stem cells were successfully isolated and used to regenerate hair follicles with two different methods.

GDNF helps grow hair and heal skin wounds by acting on hair stem cells.

Skin stem cells are maintained by signals from nearby cells and vary in their ability to renew and mature.

Fat-related cells are important for initiating hair growth.

Aging mice have slower hair regeneration due to changes in signal balance, but the environment, not stem cell loss, controls this, suggesting treatments could focus on environmental factors.

Melanocyte development from neural crest cells is complex and influenced by many factors, and better understanding could help treat skin disorders.

Pitx2 helps outer root sheath cells differentiate but can't start hair growth on its own.

Stem cells have great potential for improving wound healing, but more research is needed to find the best types and ways to use them.

KLF4 is important for maintaining skin stem cells and helps heal wounds.

The document concludes that while "hair follicle cloning" shows promise for unlimited donor hair, it faces challenges with consistency and safety in humans.

Bioluminescence imaging can track hair follicle cells and help study hair regrowth.

Hair follicle stem cells are important for maintaining healthy skin and interact with many signals.

The document concludes that specific cells are essential for hair growth and more research is needed to understand how to maintain their hair-inducing properties.

A Gsdma3 mutation causes hair loss due to stem cell damage from skin inflammation.

New hair can grow from large wounds in mice, but less so as they age, involving reprogramming of skin cells and specific molecular pathways.

Male hair loss is caused by inactive hair follicle stem cells.

Wnt/ß-catenin signaling is crucial for regulating skin stem cells and hair growth, with the right levels and timing needed for proper function.

MicroRNAs are important for hair growth regulation, with Dicer being crucial and Tarbp2 less significant.

Old people have less hair because their hair follicles don't regenerate as well, not because of fewer stem cells, and a protein called follistatin might help reactivate hair growth.

HAP stem cells in hair follicles could help repair nerves and spinal cords.

ALRN-6924 may prevent hair loss caused by chemotherapy.

Mice can grow new hair follicles after skin wounds through a process not involving existing hair stem cells, but requiring more research to understand fully.

Gamma rays did not change hair follicle density but increased white and hypopigmented hairs in mice.

Hair can regrow in adult mice's skin after injury, and this regrowth doesn't come from existing hair cells but from skin cells in the wound, with Wnt7a protein helping this process. This could help treat baldness and scarring.