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Skin spheroids with both outer and inner layers are key for regrowing skin patterns and hair.

Normal skin cell renewal doesn't need RAR signaling, but vitamin A-related skin thickening does.

Skin stem cells are maintained by signals from nearby cells and vary in their ability to renew and mature.

Skin changes help detect graft-versus-host reaction early after bone marrow transplants.

Retinoic acid affects skin and hair health by working with specific receptors, and its absence can lead to hair loss and skin changes.

New research suggests that skin cell renewal may not require a special type of cell previously thought to be essential.

Certain skin cells near the base of hair muscles may help renew and stabilize skin, possibly affecting skin disorder understanding.

The we/we wal/wal mice have defects in hair growth and skin layer formation, causing hair loss, useful for understanding alopecia.

Removing centrosomes from skin cells leads to thinner skin and stops hair growth, but does not greatly affect skin cell differentiation.

Skin stem cells maintain and repair the outer layer of skin, with some types being essential for healing wounds.

CD4+ skin cells may be precursors to basal cell carcinoma.

Skin development in mammals is controlled by key proteins and signals from underlying cells, involving stem cells for maintenance and repair.

Skin structure complexity and variability are crucial for assessing skin toxicity in safety tests.

The study found that mouse sweat glands develop before birth, mature after birth, and have specific keratin patterns.

The PP2A-B55α protein is essential for brain and skin development in embryos.

Vitiligo causes white skin patches and is linked to autoimmune issues.

The skin and mucous membranes can regenerate over the basement membrane after damage, using nearby surviving cells.

The document concludes that adult mammalian skin contains multiple stem cell populations with specific markers, important for understanding skin regeneration and related conditions.

Basal cell carcinomas have much higher levels of Vitamin D3 receptors compared to healthy skin.

ATP-dependent chromatin remodeling is crucial for skin development and stem cell function.

Blocking TSLP reduces skin inflammation and cell overgrowth in psoriasis.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

Certain transcription factors are key in controlling skin stem cell behavior and could impact future treatments for skin repair and hair loss.

Changes in keratin affect skin health and can lead to skin disorders like blistering diseases and psoriasis.

Different types of stem cells and their environments are key to skin repair and maintenance.

Mirex seems to promote a unique group of skin cells different from those affected by another tumor promoter, TPA.

Vitamin D receptor is crucial for skin wound healing.

The method increases stem-like cells for better skin regeneration.

Epidermal stem cells could lead to new treatments for skin and hair disorders.

Immune cells are essential for early hair and skin development and healing.