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DHT affects bone growth by altering gene activity in osteoblasts, potentially complicating steroid use.

Anti-androgen drugs can treat conditions like prostate cancer and male pattern baldness by blocking effects of testosterone and DHT.

Researchers developed new compounds that target the androgen receptor effectively with fewer side effects.

Higher androgen levels and site-specific AR expression cause sex-related skeletal differences, and certain steroids can boost AR expression and androgen effects in bone cells.

A three-month treatment with a GnRH agonist significantly lowered androgen levels and 5α-reductase activity in men with benign prostatic hyperplasia.

Prostate cells have proteins that bind to specific hormones, which can increase protein production when activated by these hormones.

Two enzymes regulate androgen receptor activity, affecting treatments for androgen insufficiency and benign prostatic hyperplasia.

Androgens increase muscle mass by promoting myoblast growth through ornithine decarboxylase.

Androgen self-administration might be controlled by membrane receptors, not nuclear ones.

Finasteride, a hair loss drug, may cause long-term sexual side effects due to changes in hormone receptor levels.

Androgens cause oil-producing skin cells with androgen receptors to mature and produce more oil.

Androgen receptor signaling causes early aging of cells important for hair growth by damaging their DNA.

Human keratinocytes do not naturally respond to androgens.

Male rats have androgen receptors in their liver, which are different from proteins that bind estrogen.

Finasteride may improve prostate cancer treatment outcomes.

Bisphenol-A (BPA) increases connections between brain cells and boosts their activity, but it blocks the effects of a male hormone on brain cell plasticity.

New steroids were effective in blocking male hormone receptors in hamster prostates.

The mouse model suggests male pattern baldness may be due to an enzyme increasing DHT and higher androgen receptor levels in hair follicles.

RU58841, a substance from France, can potentially block the effects of hormones that cause hair loss and excessive hair growth, performing better than a similar substance, cyproterone acetate.

Blocking glucocorticoid receptors improves glucose metabolism in a PCOS mouse model.

The conclusion is that a treatment called cp-asiAR can reduce hair loss and promote hair growth, making it a potential new therapy for androgenetic alopecia.

A 12-year-old boy with PAIS successfully developed male characteristics using high-dose testosterone and anastrozole.

Male hormones can decrease the ability of immune cells to fight bacteria.

cp-asiAR may effectively treat hair loss by targeting androgen receptors.

Higher levels of heat shock protein 27 and lower levels of miR-1 can increase AR levels, leading to hair loss in men.

cp-asiAR may effectively treat androgenetic alopecia by promoting hair growth and reducing androgen receptor activity.

AGA causes hair loss by shrinking hair follicles due to DHT binding, and can be treated with finasteride and minoxidil.

Mutations at Val-889 and Arg-752 disrupt key interactions in the androgen receptor, affecting its function.

Cells from balding scalps have more androgen receptors than cells from non-balding scalps.

DHT in the human prostate is produced through multiple pathways, not just from testosterone.