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Skin stem cells maintain and repair the outer layer of skin, with some types being essential for healing wounds.

The 3D scaffold helped maintain hair cell traits and could improve hair loss treatments.

Human scalp fat stem cells showed improved cartilage-like development on a special scaffold with freeze-thaw treatment.

Cathepsin L is essential for normal hair growth and development.

Wnt signaling is crucial for skin wound healing and reducing scars.

The protein Par3 is crucial for healthy skin, affecting the skin barrier, cell differentiation, and stem cell maintenance.

Activating ER-β, not ER-α, improves skin cell growth and wound healing.

In vivo lineage labelling is better than in vitro methods for identifying and understanding stem cells.

The study found new details about human hair growth and suggests that preventing a specific biological pathway could potentially treat hair graying.

Rat whisker cells can help turn other cells into nerve cells and might be used to treat brain injuries or diseases.

SHISA6 helps maintain certain stem cells in mouse testes by blocking signals that would otherwise cause them to differentiate.

Androgens prevent hair growth by changing Wnt signals in cells.

Adult esophageal cells can start to become like skin cells, with a key pathway influencing this change.

Signals from skin cells controlled by Rac proteins help turn certain precursor cells into white fat cells.

Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

Super-enhancers controlled by pioneer factors like SOX9 are crucial for stem cell adaptability and identity.

Key genes are crucial for Drosophila wing development and could be insecticide targets.

The nude gene causes skin cell overgrowth and improper development, leading to hair and urinary issues.

Exosomes from dermal papilla cells help hair growth by making hair follicle stem cells multiply and change.

Activating β-catenin increases melanocytes and decreases Schwann cells.

Hair follicles are complex, dynamic mini-organs that help us understand cell growth, death, migration, and differentiation, as well as tissue regeneration and tumor biology.

The conclusion is that the nuclear lamina and LINC complex in skin cells respond to mechanical signals, affecting gene expression and cell differentiation, which is important for skin health and can impact skin diseases.

The document concludes that careful design of genetic fate mapping experiments is crucial for accurate cell lineage tracing in mice.

Stem cells help heal skin wounds by moving and changing roles, working with other cells, and needing more research on their activation and behavior.

Sweat glands and hair follicles are determined by opposing signals, with BMPs promoting sweat glands and blocking BMPs leading to hair follicles.

Certain mature cells in mouse lungs can turn back into stem cells to aid in tissue repair.

Jumonji genes are important for development and their mutations can cause abnormalities, especially in the heart and brain.

Epidermal Wnt/β-catenin signaling controls fat cell formation and hair growth.

Different skin stem cells help heal wounds, with hair follicle cells becoming more important over time.

The p53 protein has complex, sometimes contradictory functions, including tumor suppression and promoting cell survival.